Answer:
The genotypes of the rooster and the chicken are homozygous and that of their offspring is heterozygous.
This case is called codominance, where the offspring receives an allele from each parent, from the rooster and the hen, because there is codominance, so neither allele is recessive and the phenotype of both alleles is expressed so the phenotype of the offspring is checkered.
We can say then that the chicken and the rooster have equal strength between their alleles. in the cases of codominance the laws of mendel do not apply.
Matter is anything that has mass and occupies space. There are three different states of matter- solid, liquid, and gas.
Answer: A unicellular organism consists of 1 single egg, and a multicelluar organism consists of 2 or more eggs.
Explanation:
Answer:
C
Explanation:
Trees take up carbon dioxide from the atmosphere and reduce to make carbohydrates. This is powered by the energy of the sun that is taped by chlorophyll pigments during photosynthesis. The energy is used to split water molecules into H+ and O2-. Then the following chemical reaction ensues;
6CO2 + 6H2O ------> C6H12O6 + 6O2
Answer:
PFFT this might help? sorry if not mate
Explanation:
Cell cycle checkpoint controls play a major role in preventing the development of cancer [see Sherr, 1994, for a more detailed discussion]. Major checkpoints occur at the G1 to S phase transition and at the G2 to M phase transitions. Cancer is a genetic disease that arises from defects in growth-promoting oncogenes and growth-suppressing tumor suppressor genes. The p53 tumor suppressor protein plays a role in both the G1/S phase and G2/M phase checkpoints. The mechanism for this activity at the G1/S phase checkpoint is well understood, but its mechanism of action at the G2/M phase checkpoint remains to be elucidated. The p53 protein is thought to prevent chromosomal replication specifically during the cell cycle if DNA damage is present. In addition, p53 can induce a type of programmed cell death, or apoptosis, under certain circumstances. The general goal of p53 appears to be the prevention of cell propagation if mutations are present. The p53 protein acts as a transcription factor by binding to certain specific genes and regulating their expression. One of these, WAF1 or Cip1, is activated by p53 and is an essential downstream mediator of p53-dependent G1/S phase checkpoint control. The function of p53 can be suppressed by another gene, MDM2, which is overexpressed in certain tumorigenic mouse cells and binds to p53 protein, thus inhibiting its transcriptional activation function. Other cellular proteins have been found to bind to p53, but the significance of the associations is not completely understood in all cases. The large number of human cancers in which the p53 gene is altered makes this gene a good candidate for cancer screening approaches.
