Answer:
In Java
import java.util.Scanner;
public class Main {
public static void main(String[] args) {
int mnth, yr;
Scanner input = new Scanner(System.in);
System.out.print("Month: "); mnth = input.nextInt();
System.out.print("Year: "); yr = input.nextInt();
boolean lpYear = (yr % 4 == 0 && yr % 100 != 0) || (yr % 400 == 0);
if(mnth == 1 || mnth == 3 || mnth == 5 || mnth== 7 || mnth == 8 || mnth == 10 || mnth == 12){
System.out.print("31 days"); }
else if(mnth == 2){
System.out.print(((lpYear) ? "29 days" : "28 days")); }
else if(mnth == 9 || mnth == 6 || mnth == 4 || mnth== 11){
System.out.print("30 days"); }
else{ System.out.print("Invalid"); }
}
}
Explanation:
See attachment for complete program where comments were used as explanation.
Answer:
Encrypt this binary string into ciphertext: 110000. Include
in your answer the formula the decoder would use to decrypt your cipher text in the format (coded answer) x N mod (m) = Y
-Decrypt this ciphertext into a binary string: 106
Now that you can see how public and private keys work in a real-world scenario, please explains how it works for computers and internet communications in relation to cybersecurity.
Explanation: ……………..
Answer:
The first IBM PC virus in the "wild" was a boot sector virus dubbed (c)Brain, created in 1986 by Amjad Farooq Alvi and Basit Farooq Alvi in Lahore, Pakistan, reportedly to deter unauthorized copying of the software they had written. The first virus to specifically target Microsoft Windows, WinVir was discovered in April 1992, two years after the release of Windows 3.0.
Explanation:
HOPE THIS HELPS
The answer is B to the question
Answer:The allosteric inhibitors of integrase (termed ALLINIs) interfere with HIV replication by binding to the viral-encoded integrase (IN) protein. Surprisingly, ALLINIs interfere not with DNA integration but with viral particle assembly late during HIV replication. To investigate the ALLINI inhibitory mechanism, we crystallized full-length HIV-1 IN bound to the ALLINI GSK1264 and determined the structure of the complex at 4.4 Å resolution. The structure shows GSK1264 buried between the IN C-terminal domain (CTD) and the catalytic core domain. In the crystal lattice, the interacting domains are contributed by two different dimers so that IN forms an open polymer mediated by inhibitor-bridged contacts; the N-terminal domains do not participate and are structurally disordered. Engineered amino acid substitutions at the inhibitor interface blocked ALLINI-induced multimerization. HIV escape mutants with reduced sensitivity to ALLINIs commonly altered amino acids at or near the inhibitor-bound interface, and these substitutions also diminished IN multimerization.
Explanation:
