Answer:
The answer is A a loss of surfactant.
Explanation:
The surfactant is a liquid that intervenes in the capillary-alveolus interface favoring the exchange of gases (oxygen, CO2) at this pulmonary level. For this reason, damage to type II pneumocytes will produce an alteration of this exchange.
Answer:
B. 28
Explanation:
24 h/ 6h= 4 times
1 day -> 4 capsules
7 days -> x
x= (7 days*4 capsules)/ 1 day
x= 28 capsules
Answer:
Thalamus to amygdala.
Explanation:
Neural pathway is the way by which signals are transmitted in the body from one region to another region. These neurons are connected by the bundle of axons or by the single axon.
The fear response and all the other response is directly or indirectly controlled by the nervous system. The fear response may take time because the information needs to travel from the thalamus region to the amygdala region of the brain.
Thus, the answer is thalamus to amygdala.
The EMT would recognize <u>compensated respiratory distress</u>.
Compensated respiratory acidosis is typically the result of a chronic condition, the slow nature of onset giving the kidneys time to compensate. Common causes of respiratory acidosis include hypoventilation due to respiratory depression (sedatives, narcotics, CVA, etc.)
Respiratory alkalosis occurs when high levels of carbon dioxide disrupt the blood's acid-base balance. It often occurs in people who experience rapid, uncontrollable breathing (hyperventilation).
Retention of bicarbonate by renal tubules to minimize the effect on blood pH of carbon dioxide retention by the lungs, such as occurs in patients with hypoventilation.
To learn more about compensated respiratory distress, here
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