Answer:
A. Children report more cognitive symptoms than adults, is true regarding panic disorders in children.
Explanation:
A. Children report more cognitive symptoms than adults.
Children with panic disorder are more anxious as compared to adults even when they are not having panic attacks. They report severe cognitive symptoms such as fear of an attack, reluctance to perform well in school, avoiding certain places due to fear of attack called agoraphobia, depression, substance abuse and suicidal behaviors. Hence, children may also need psychotherapy along with panic disorder medications so that they could control their anxiety. Early treatment of panic disorder with cognitive behavioral therapies may prevent complications such as agarophobia, and depression in children.
B. Children experience only cued panic attacks.
Both children and adolescents have unexpected panic attacks which might be triggered by anything which causes anxiety in them. They should have varying periods of fear and discomfort which might last minutes to hours.
C. Children may report a general fear of becoming sick rather than specific physical symptom
A child suffering from panic attacks may have heart-attack like symptoms with a racing heart, fear of death and losing control, obsessive-compulsive symptoms, and physical symptoms including nausea, vomiting, dizziness, shortness of breath etc. Moreover, children are also left with an intense fear of another panic attack which might trigger these symptoms. Thus, they become less functional and has a fear of changing places, habits as anything might trigger their symptoms. So, children are more anxious about the physical symptoms ensuing the panic attack and tries their best to avoid it.
MCV = Hct × 10/RBC (84-96 fL) •Mean corpuscular Hb (MCH) = Hb × 10/RBC (26-36 pg) •Mean corpuscular Hb concentration (MCHC) = Hb × 10/Hct (32-36%) A rapid method of determining whether cellular indices are normocytic and normochromic is to multiply the RBC and Hb by 3.
Answer:
The answer to the question: Electrical impulses or action potentials (AP) cannot propagate across a synaptic cleft. Instead, neurotransmitters are used to communicate at the synapse, and re-restablish the AP in the post-synaptic cell, would be, true.
Explanation:
Essentially, although the CNS, PNS and ANS (Central Nervous System, Peripheral Nervous System and Autonomic Nervous System) are electrical systems, that use the electricity generated through action potentials, that in turn, are created by the exchange of electrically charged ions from within and without the neuronal cells, these electrical impulses do not pass the pre-synaptic, and post-synaptic cleft. Instead, once an AP has reached the terminal end of the pre-synaptic neuron, neurotransmitters are released by this one, into the cleft, which are then taken up by the receptors present in the post-synaptic neuron. Depending on the type of transmitter released, there will be an inhibitory or excitatory effect. Then, with the transmitter attached, the post-synaptic neuron will depolarize and a new AP will be generated in the post-synaptic neuron, which will carry on. Messages, thus are transmitted that way between neurons, and also, between the nervous system and the organs they influence.
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The principal pathologic finding in the diagnosis of bronchiectasis is chronic dilation of the airways (bronchi and bronchioles).
<h3>What are the airways?</h3>
The airways are the structures that the body uses to interchange gases with the surrounding environment.
The bronchi are major airways, whereas bronchioles are the smallest structures that generate tiny air sacs known as alveoli.
Moreover, bronchiectasis symptoms include, among others, chest pain, wheezing and shortness of breath.
Learn more about the airways here:
brainly.com/question/4351510
