Jayden, you didn’t put the diagram so we can’t help. ｡ﾟ(ﾟ´Д｀ﾟ)ﾟ｡
Answer:
Crossing over increases the genetic variation
Errors in crossover formation result in chromosomal rearrangements (duplications and deletions) and may also cause chromosome nondisjunction
Explanation:
Crossing over, also known as recombination, refers to the exchange of genetic material (DNA) between non-sister chromatids of homologous chromosomes during meiosis. Crossing over increases the genetic variation of the resulting gametes, allowing different genetic combinations to be passed from parents to offspring. Unequal crossing over is a type of error associated with chromosomal duplication and/or deletion events. Unequal crossing over occurs when homologous sequences are not paired precisely. Moreover, errors in crossing over may also cause chromosome nondisjunction, thereby leading to aneuploidies.
AraC protein certain to the ara
operator works as a repressor that avoid transcription of the same ara operon
and the araC gene AraC protein is an activator in the presence of arabinose and
repressor in the absence of arabinose -With arabinose absent, the AraC protein
stays as
a homodimer. One AraC protein put together to the ara initiator and the other
protein now binds to a different regulatory site in DNA called the ara operator.
Answer:
The loading dose of N-acetylcysteine is 8,700 mg (150 mg per 55 kilograms).
The complete cycle of treatment with N-acetylcysteine comprises 3 consecutive intravenous infusions, distributed as follows:
1 infusion: Initial dose of 150mg / kg of body weight in 200 ml of solution to pass in one hour.
2 infusion: 50 mg / k of weight in 500 ml of solution to pass in 4 hours
3 infusion: 100mg / kg of weight in 1000ml to pass in the following 16 hours.
