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Explanation:
when it's fully charged it's a ATP and when it's runs down it's ADP
and there are times when the cells need more energy and splits off another phosphate so it goes from ADP adenoside di-phosphate, to AMP, adenosine mono-phosphate
and there you have it
Answer:
1. Inhibiting IP3 channels, leading to decreased Ca2 in the sarcoplasm and reduced contraction.
2. Increasing the relative activity of MLCP, leading to a decrease in tension.
3. Activating K channels, increasing K leaking out of the cell which hyperpolarizes it and decreases the likelihood of Ca2 entry.
Explanation
In smooth muscle, cyclic AMP (cAMP) mediates relaxation because cAMP inhibits a specific kinase required for myosin light chain protein (MLCP) phosphorylation, thereby triggering contraction in the smooth muscles. It has been shown that cAMP inhibits 1,4,5-trisphosphate (IP3)-dependent calcium ions (Ca 2+) release by activation of the cGMP-dependent protein kinase (PKG). PKG proteins act to modulate Ca2+ oscillations by stimulating sarcoplasmic Ca2+-ATPase membrane proteins, increasing Ca2+ in the sarcoplasmic reticulum stores and Ca2+ efflux from the cells, and activate voltage-gated potassium (K) channels, thereby leading to membrane hyperpolarization and reducing Ca2+ entry through Ca2+ channels.
Answer:
The prolonged electrical depolarization of cardiac muscle cells -that occurs during contraction- is due primarily to the persistent influx of calcium ion
Explanation:
The action potential of the heart muscle is longer with respect to skeletal muscle (around 300 milliseconds), and this is due to the activity of calcium (Ca⁺⁺ ) in the intracellular compartment.
The initial depolarization of cardiac muscle fiber depends on the entry of sodium (Na⁺) into the cell. However, for the action potential to occur and be maintained, Ca⁺⁺ must increase its cytoplasmic levels, which depends on:
- The increase in intracellular sodium induces the release of Ca⁺⁺ from the sarcoplasmic reticulum.
- Calcium entry from the extracellular space through the voltage dependent Ca⁺⁺ channels.
- The entry of extracellular Ca⁺⁺ causes the release of more Ca⁺⁺ ions by the sarcoplasmic reticulum, further increasing its intracellular concentration.
This is how the ion that guarantees the duration of the action potential of the cardiac muscle cell is the Ca⁺⁺.
Learn more:
Calcium, sodium and cardiac muscle cells brainly.com/question/4473795
