The answer would be CODOMINANCE because codominance is when 2 genotypes show up at the same time on an object, where incomplete dominance is when 2 different genotypes combine and create a whole new phenotype
Because most cigar and pipe smokers do not<u> </u><u>inhale the smoke</u> , as a group they have a lower risk of cancer than cigarette smokers.
The cigarettes, cigars, and pipe tobacco are made out of dried tobacco leaves. The other substances are usually added for flavor and to make smoking more gratifying. The smoke from these products is a complex mixture of chemicals produced by flaring tobacco and its additives.
There are at least 70 known chemicals to cause cancer. These cancer-causing chemicals are referred to as carcinogens.
It is so because cigars and pipes are usually believed to be less harmful way than to smoke tobacco. It was once a trend to use cigars in the 1990s, luring/dragging the young and the old.
Most of the people think cigars are less harmful to their health, but they actually pose the same risk for oral cancer as cigarettes do. Many cigar smokers don't inhale the smoke, but still the risk for oral, throat, and esophageal cancer is alike as for cigarette smokers.
To learn more about carcinogens here
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Answer:
Normal Strand: alanine - methionine - histidine
Mutated Strand: glutamine - cysteine - no third amino acid.
Explanation:
<h3>mRNA Structure</h3>
Messenger ribonucleic acid (mRNA) is the RNA that is used in cells for protein synthesis. It has a single strand made by the transcription of DNA by RNA polymerase. It contains four nucleotides: Adenine (A), Guanine (G), Cytosine (C), and Uracil (U).
<h3>DNA Replication</h3>
Before transcribing, we need to create the complementary strand of the DNA. We're going to write out the nucleotides of the complementary strand by matching the nucleotides in these pairs: (A & T) and (C & G).
Normal Strand: GCA ATG CAC
Complementary Strand: CGT TAC GTG
Next, we can transcribe this to find our mRNA. We're going to do the same thing to the complementary DNA strand, but with Uracils instead of Thymines. So our pairs are: (A & U) and (C & G)
Complementary DNA Strand: CGT TAC GTG
mRNA Strand: GCA AUG CAC
You'll notice that the mRNA strand is almost exactly like the new mRNA strand, but with Uracil instead of Thymine.
<h3>Reading Codons</h3>
Each set of three nucleotides is known as a codon, which encodes the amino acids that ribosomes make into proteins. To read the codons, you need to have a chart like the one I attached. Start in the middle and work your way to the edge of the circle. Some amino acids have multiple codons. There are also "stop" and "start" codons that signify the beginning and ends of proteins.
mRNA Strand: GCA AUG CAC
Amino Acids: Ala Met His
Our sequence is alanine, methionine, and histidine.
<h3>Frameshift Mutations</h3>
A frameshift mutation occurs when a nucleotide is either added or removed from the DNA. It causes your reading frame to shift and will mess up every codon past where the mutation was. This is different than a point mutation, where a nucleotide is <em>swapped</em> because that will only mess up the one codon that it happened in. Frameshift mutations are usually more detrimental than point mutations because they cause wider spread damage.
<h3>Mutated Strand</h3>
Let's repeat what we did earlier on the mutated strand to see what changed.
Mutated Strand: CAA TGC AC
Complementary Strand: GTT ACG TG
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Complementary DNA Strand: GTT ACG TG
mRNA Strand: CAA UGC AC
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mRNA Strand: CAA UGC AC
Amino Acids: Glu Cys X
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Our amino acid sequence is glutamine, cysteine, and no third amino acid.
As you can see, removing the first nucleotide of the strand caused every codon to change. The last codon is now incomplete and won't be read at all. If this happened in a cell, the protein that was created from this mutated strand would be incorrect and may not function completely or at all.
Answer:srry, i dont know the answer
Answer:
A) The radial orientation of cellulose microfibrils in the cell walls of guard cells causes them to bow outward and open when the stomatal pore when turgid.
Explanation:
Stomata are pores on the surface of leaves that work as valves controlling the gas exchange in plants; they are bordered by a pair of curved guarded cells in charge of opening and closing by increasing or decreasing the stomatal pore area.
Guard cells walls structure consist of a combination of stiff cellulose microfibrils oriented along the circumference of the cells and a softer matrix material which makes it possible for stomata pore to open by increasing water pressure (turgor).
Option B states that when guard cells become turgid, they close the stomata. This is a false statement as we explained before.
Option C states that light (known stimulant for opening stomata) causes guard cells to become flaccid which is also false. Guard cells become turgid in order to open the stomatal pore.
Option A is correct.