When a DNA sequence alteration results in a stop codon rather than a codon that specifies an amino acid, it is known as a nonsense mutation. This is rarely seen in 10% of patients with genetic disease.
<h3>What is nonsense mutation?</h3>
A nonsense mutation in a DNA sequence causes a premature stop codon, also known as a nonsense codon, in the transcribed mRNA as well as a shortened, ineffective, and typically nonfunctional protein product.
Because stop codons, also known as nonsense codons, signal the completion of protein synthesis rather than encoding for an amino acid, they are the source of the term "nonsense mutation."
Examples of illnesses for which nonsense mutations have been implicated as contributing factors include: Cystic fibrosis (produced by the G542X mutation in the cystic fibrosis transmembrane conductance regulator); (CFTR) Beta-globin (thalassemia) Hurler disease.
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<span>(1) Does the pH of water affect the growth of radish plants?
</span><span>(2) pH of the water
</span><span>(1) repeating the experiment several times</span>
Histone deacetylase is responsible for removing the acetyl group from the histone 3 lysine 9 residue. Remember that deacetylation is one step in converting euchromatin to heterochromatin. Because euchromatin is transcriptionally active (transcriptional machinery is able to reach gene of interest), and blocking histone deacetylase activity would result in an the DNA remaining as euchromatin, we would expect to see an increase in transcriptional activity.
So there’s your answer: An increase in transcriptional activity.
Answer:
Pneumocystosis
Explanation:
Pneumocystosis is an infection of the lungs. Its causative organism is the microorganism Pneumocystis carinii. Pneumocystosis is nearly entirely and completely observed in individuals that have a immune systems that have been weakened and diminished by AIDS or chemotherapy. Pneumocystosis is majorly a terminal occurrence in AIDS patients.
