Answer:
INTERNEUTRONS
Explanation:
INTERNEUTRONS
Interneutrons are really the highest number of neurons and spinal cord. They are accountable for the diverse communication system of the nervous system and try and resolve between inputs and outputs.
Interneurons are groups of nerve cells commonly found in nervous system integrative areas where axons & dendrites are restricted to a specific area of brain.
I believe its two <span>PGAL a0 molecules.</span>
Answer:
a. + glucose, + lactose = On
b. - glucose, - lactose = Off
c. + glucose, - lactose = Off
d. - glucose, + lactose = On
Explanation:
Lac operon has both types of control, repressible and inducible.
Whenever glucose level is low in the cell, an enzyme known as adenylyl cyclase raises the level of cAMP which forms a dimer with CRP protein and they both act as activator of lac operon and cause expression.
Apart from this, when lactose is present in the cell, β-galactosidase enzyme metabolizes lactose to form allolactose which causes allosteric repulsion in the lac repressor and causes its removal from the operator. As soon as repressor is removed lac operon gets activated.
In wild type lac operons, the expression of lac operon occurs when glucose level is low in the cell and lactose is present but in this mutant presence or absence of glucose will not make a difference because CRP will bind Plac promoter independent of cAMP level i.e. activator CRP will work even in high glucose concentration. If lactose is present then lac operon will always express so in option 'a & d' lac operon will express but in option 'b & c' it will not express.
Answer:
cause it's obvious that you dumb bruh lol
Answer:
Neutrophils help fight infections because they ingest microorganisms and secrete enzymes that destroy them. A neutrophil is a type of white blood cell, a type of granulocyte and a type of phagocyte.
Explanation:
Neutrophils display adhesion glycoproteins on their surface to bind endothelial and subendothelial structures. They move randomly until they find a damaged site. Unless neutrophils are activated, endothelial cells do not tend to adhere. When inflammation mediators (IL-1, FNT) activate endothelial cells, they express P-selectin and E-selectin on the surface. The expression of glycoproteins and L-selectin cause the initial adhesion of the non-stimulated neutrophil to the activated endothelium, slowing it down by rolling it over the endothelium. Activated endothelial cells, opsonized particles, immune complexes, FEC-G, FEC-GM and chemoattractants produce factors that stimulate neutrophil activation. Expressing β2 integrin (endothelium adhesion molecule) Neutrophils expand and form pseudopods. Neutrophil activation also promotes degranulation, superoxide generation, and arachidonate metabolite production.
