Answer:
I think it's C but I might be wrong so I apologize if I am.
Answer:
Option-C
Explanation:
A pulse-chase experiment which is used to study the cellular process taking place in the cells. This uses the labelled molecule which is incorporated into the cellular pathway and then the unlabeled molecules are used over time to determine how much time is required by the unlabeled molecule to replace the labelled molecule.
In the given question, the pancreatic cell which secretes the digestive enzyme known as the acinar cell is labelled with the leucine and then used unlabeled leucine.
When observed after three hours, the black spots will appear on the exterior of the cell as the protein has been synthesised by the ER and now has been secreted from the cell.
Thus, Option-C is the correct answer.
The mutation resulting in sickle cell disease changes one base pair of DNA so that a codon now codes for a different amino acid, making it an example of a missense mutation. Missense mutation is the mutation characterized with <span>changes in one base pair so that the whole amino acid is changed.</span>
Answer:
Antidiuretic hormone
Explanation:
Antidiuretic hormone that is synthesized and produced in the brain by the hypothalamus, is secreted from the pituitary gland, where it is stored. This hormone is released into the blood stream in response to changes in the water balance in the blood. This hormone helps the kidneys to control and regulate the amount of water in the blood
Answer:
Mitochondrial Disorder
Explanation:
The LHON stands for leber hereditary optic neuropathy is a type of mitochondrial disorder. The disorder is generally observed in young males. The mitochondrial disorder are transferred from mother to its progeny. The disorder causes retinal ganglion cells (RGCs) and the exons of RGCs to degenerate. The disorder causes sudden painless failure of vision. The disorder leads to loss of central vision, optic atropy and central scotoma.
The most of the individuals with LHON do not possess the signs of the disorder. The disorder is caused by mutation in maternal DNA, thus it is a type of mitochondrial disorder.
