Explanation:
Many mRNAs have sequences of 50 - 150 nt rich in A and U in the 3' untranslated region. These sequences, called AU-rich elements, are important to regulate mRNA stability, because they bind proteins that target the mRNA for degradation.
When a protein binds to the AU-rich element of the mRNA, the polyA tail can be shortened and/or the 5'cap is removed, promoting mRNA degradation. In addition, the AU-rich elements in some mRNAs have been shown to inhibit protein translation.
The mRNA of the overexpressing cell line is about 200nt shorter than the normal cell line; this could mean that it has a deletion in the 3'UTR comprising an AU-rich element. If this is missing, the mRNA will have an extended half-life (because it won't be targeted for degradation) or it will be translated more. As a result, the protein expression will be increased.
carbon's atomic number is =6
oxygen's atomic number is=8
potassium's atomic number is=19
arsenic's atomic number is=33
lodine atomic number is=53
calcium's atomic number is=20
boron's atomic number is=5
neon's atomic number is=10
protons
carbon=6
oxygen=8
potassium=19
arsenic=33
lodine=53
calcium=20
boron=5
neon=10
electrons
carbon=6
oxygen=8
potassium=19
arsenic=33
lodine=53
calcium=20
boron=5
neon=10
groups of the elements
carbon group 6
oxygen group 14
potassium group 1
arsenic group 15
lodine group 17
calcium group 2
boron group 5
neon group 18
valence electron
carbon 2;4
oxygen 2
potassium 1
calcium 2
boron 3
neon 0
lodine 1
cation or anion
c-
o+
k+
ca+
l-
b3+
ne+
as+
Answer:
The main product of photosynthesis is glucose
Hope that helped and also I will change my profile to have something different than the sonic movie because ugh it's weird
Prader-Willi syndrome (PWS) is a gentic disorder which has an impact on numerous physiological systems. PWS affected individuals (specifically babies) experience delayed growth, significant hypotonia (low muscle tone), and feeding issues. It does effect circadian rhythms in mice models.
SNORD116, often referred to as HBII-85, is a non-coding RNA (ncRNA) molecule that contributes to the alteration of other small nuclear RNAs (snRNAs). Unlike the majority of other snoRNAs, SNORD116 is not significantly complementary to ribosomal RNA and is expressed widely in the brain (but not in PWS patients).
According to the studies, SNORD116 cause sleep defect in patients with Prader-Willi syndrome. Same observation was seen in mouse models too. Paternal expression of SNORD116 is thought to be a potential gene for the sleep disruptions/circadian rhythm’s that the majority of PWS sufferers.
To learn more about circadian rhythm click here
brainly.com/question/6434404
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