Answer:
Having considered how an appropriate primary immune response is mounted to pathogens in both the peripheral lymphoid system and the mucosa-associated lymphoid tissues, we now turn to immunological memory, which is a feature of both compartments. Perhaps the most important consequence of an adaptive immune response is the establishment of a state of immunological memory. Immunological memory is the ability of the immune system to respond more rapidly and effectively to pathogens that have been encountered previously, and reflects the preexistence of a clonally expanded population of antigen-specific lymphocytes. Memory responses, which are called secondary, tertiary, and so on, depending on the number of exposures to antigen, also differ qualitatively from primary responses. This is particularly clear in the case of the antibody response, where the characteristics of antibodies produced in secondary and subsequent responses are distinct from those produced in the primary response to the same antigen. Memory T-cell responses have been harder to study, but can also be distinguished from the responses of naive or effector T cells. The principal focus of this section will be the altered character of memory responses, although we will also discuss emerging explanations of how immunological memory persists after exposure to antigen. A long-standing debate about whether specific memory is maintained by distinct populations of long-lived memory cells that can persist without residual antigen, or by lymphocytes that are under perpetual stimulation by residual antigen, appears to have been settled in favor of the former hypothesis.
The answer is A.True but they are not the only ways in which this can occur.
Abundant amount of amino acids made up a polypeptide
The answer is four haploid cells
Answer: diabetes mellitus
Explanation:
This is a disease condition associated with elevated blood glucose levels.
It is due to abnormalities of the Pancreas. The pancreas coordinates two hormones INSULIN AND GLUCAGON in its cells.
INSULIN converts excess sugar to GLYCOGEN while GLUCAGON breaks down GLYCOGEN to sugar when blood glucose/glucose levels drops.
If the pancreas beta-cells of glucose are faulty, INSULIN fails to converts excess glucose to glycogen; therefore glucose builds up in the blood plasma,and failed to enter the needed cells. Large amount of glucose is wasted in urine.This is diabetes Mellitus.
The two types of diabetes are:
Type 1 and Type 2.