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ollegr [7]
3 years ago
10

Why is still water an ideal environment for the formation of mold and cast fossils?

Biology
2 answers:
lyudmila [28]3 years ago
5 0

Answer:

Fossil of the hard-shelled organism which shows the surface contours giving the exact 3D shape of the organism is called a mold and cast fossil. Fossils are formed in water bodies where the sediments can be deposited over the dead and decayed organisms.  

When hard-shelled organism dies, still water plays important role in the formation of these fossils as: when water from sediment leaches our it decays the organic matter which creates void which is filled by sediment or mineral matter and the acidic water dissolves and breakdown the shell which gets surrounded by the mud or sand which hardens.

Thus, water plays an important role in the formation of mold and cast fossils.

Natasha_Volkova [10]3 years ago
4 0

Still waters allow for sediments to settle down on fossils without disturbance. This gives the minerals in the sediments time to perfuse in the fossils and petrify the fossil.  The sediments also settle on the fossils without disturbance, take the form of the fossil and get cemented forming molds.

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How is energy released from ATP?<br> ANSWER CHOICES IN THE PIC
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lozanna [386]

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Read 2 more answers
DCCD (dicyclohexylcarbodiimide) inhibits oxidative phosphorylation when the substrate is mitochondrial NADH. DCCD is a drug that
VARVARA [1.3K]

Answer:

A) Decreases cellular energy production

B) DCCD also affects K+ transport

Explanation:

A) Consequences are of DCCD on cellular energy production: <em>Decreases cellular energy production</em>

ATP-synthase pump is composed of two subunits: F1 catalytic subunit that synthesizes ATP, and F0 proton pumping subunits, that transport H+ through the membrane. F1 subunit might act independently of F0 to produce ATP, but this molecule can not be released without H+ gradient, which generates a movement necessary for ATP release from the catalytic center.

When any of the parts composing F0 react with DCCD, the subunit can not transport H+ through the membrane. DCCD inhibits the enzyme activity by blocking the protons´ flow.

As DCCD blocks the protons´ flow, and the protons´ flow is necessary to release the ATP molecule from the F1 subunit, no other ADP + Pi can enter to F1 subunit, and the production of ATP stops.

B) Other cellular effects of DCCD

There seem to be other effects of DCCD on cell activity, some of which are still under study. To name a few:

  • Diimide from DCCD seems to stimulate cytochrome b reduction and inhibits its reoxidation by ferricyanide.
  • When exposing the cell to high concentrations of DCCD for a long time, might occur an alteration in the electron transporting chain
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DCCD strongly inhibits the simultaneous flow of H+ and K+. First, it inhibits H+ flow, acidification of the environment stops, but at this point, K+ keeps moving through the membrane. Once the H+ flow has ceased, the K+ flow slowly decreases until it finally stops moving. There is a lag time in the DCCD effect on K+ flow to the instantaneous effect on H+ flow.

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