<span>Based on a study of recreational drugs that have been linked with a higher incidence of sudden infant death syndrome was the use of Marijuana and other recreational drugs like cocaine, crack, crystal, amphetamine, methamphetamine, and speed but cocaine was the most commonly used drug. The basis of the infant sudden death was that both maternal and paternal were using at the time of conception and during in the latter stage of pregnancy. Tobacco smoking of the mother during pregnancy can also affect the life of the baby. There is statistics base on the study on how many percents it will affect the condition of the infant.</span>
Answer:
Location of the Cell Bodies Also, the cell bodies of the preganglionic neuron are located in the brain or spinal cord while the cell bodies of the postganglionic neuron are located in the ganglion. Axons of Preganglionic and Postganglionic Neurons
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Practicing three point shots to improve free throw accuracy contradicts the SPECIFICITY principle of exercise.
The specificity principle of exercise states that, to become an expert in a particular exercise or skill one should perform that exercise or skill. That is, the things that one do during physical activity should be relevant to one desired outcomes, thus, the training regimen must go from general to specific during training.
Answer:
C) Through genomic imprinting, methylation regulates expression of the paternal copy of the gene in the brain.
Explanation:
The pattern of gene expression wherein either paternal or maternal gene is expressed in specific cells while the other one is prevented from expression is known as genomic imprinting.
In the given example, the maternal copy of the gene on chromosome 15 is expressed in brain cells while its paternal copy is not expressed in these cells. Hence, the pattern of expression of this gene is regulated through genome imprinting. One of the mechanism is methylation of cytidine residues of CpG islands of the DNA that are more frequently present within promoters of the genes.
When the cytidine residues of these sequences are methylated into 5-methylcytidine, the transcription factors do not bind to these promoters preventing the expression of these genes.
Hence, methylation of cytidine residue in CpG islands of the promoters of the gene present on chromosome 15 could have silenced its expression in brain cells.
