The answer is C
Your making an identical copy
They lay eggs, turn from tadpolls to Frogs, and support Hitler
Answer:
A. in order to release the energy found in food.
Explanation:
Every cell in your body needs oxygen to function. You get the oxygen your cells need from the air you breathe. The air you breathe is made up of 20 percent oxygen. The rest of the air is mostly nitrogen (79%). Your body cells use the oxygen you breathe to get energy from the food you eat. This process is called cellular respiration. During cellular respiration the cell uses oxygen to break down sugar. Breaking down sugar produces the energy your body needs. This is very similar to wood burning in a fire. As the wood burns, it combines with oxygen and releases heat energy and carbon dioxide. When the cell uses oxygen to break down sugar, oxygen is used, carbon dioxide is produced, and energy is released. But instead of heat energy, much of the energy produced in cellular respiration is stored chemically for the cell to use later. Carbon dioxide is the waste product of cellular respiration that you breathe out each time you breathe. Blood picks up oxygen and releases carbon dioxide in the lungs. The opposite takes place in the cells where the blood releases oxygen and picks up carbon dioxide.
Answer:
Only when a microorganism has successfully established a site of infection in the host does disease occur, and little damage will be caused unless the agent is able to spread from the original site of infection or can secrete toxins that can spread to other parts of the body. Extracellular pathogens spread by direct extension of the focus of infection through the lymphatics or the bloodstream. Usually, spread by the bloodstream occurs only after the lymphatic system has been overwhelmed by the burden of infectious agent. Obligate intracellular pathogens must spread from cell to cell; they do so either by direct transmission from one cell to the next or by release into the extracellular fluid and reinfection of both adjacent and distant cells. Many common food poisoning organisms cause pathology without spreading into the tissues. They establish a site of infection on the epithelial surface in the lumen of the gut and cause no direct pathology themselves, but they secrete toxins that cause damage either in situ or after crossing the epithelial barrier and entering the circulation.
Most infectious agents show a significant degree of host specificity, causing disease only in one or a few related species. What determines host specificity for every agent is not known, but the requirement for attachment to a particular cell-surface molecule is one critical factor. As other interactions with host cells are also commonly needed to support replication, most pathogens have a limited host range. The molecular mechanisms of host specificity comprise an area of research known as molecular pathogenesis, which falls outside the scope of this book.
While most microorganisms are repelled by innate host defenses, an initial infection, once established, generally leads to perceptible disease followed by an effective host adaptive immune response. This is initiated in the local lymphoid tissue, in response to antigens presented by dendritic cells activated during the course of the innate immune response (Fig. 10.2, third and fourth panels). Antigen-specific effector T cells and antibody-secreting B cells are generated by clonal expansion and differentiation over the course of several days, during which time the induced responses of innate immunity continue to function. Eventually, antigen-specific T cells and then antibodies are released into the blood and recruited to the site of infection (Fig. 10.2, last panel). A cure involves the clearance of extracellular infectious particles by antibodies and the clearance of intracellular residues of infection through the actions of effector T cells.
Explanation:
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