Answer:
A
Explanation:
you dont have a picture of the molecule but assuming it could be the one i'm looking at, the answer is A.
Answer:
<h2>G-A mutation is a point mutation in which one nucleotide is replaced by another nucleotide, it is an example of Transition mutation (a base substitutions that changes a purine nucleotide to another purine.</h2>
Explanation:
1. Defective Mitochondrial Gene Expression Results in Reactive Oxygen Species (ROS).
2. Elevated production of reactive oxygen species (ROS), and increased oxidative stress, results in defects in proper functioning of cell by causing mutation in the DNA and mitochondrial genes.
3. If the mutation is silent mutation, then there is no harm or less harm in the cell, if the mutation is missense or nonsense type then it can affect the functioning of the cell and phenotype of that organism.
4. If their is mutation in mitochondrial tRNA gene, it can affect the translation of mitochondrial mRNA and gene expression.
The answer is C. Hereditary means to inherit traits from your parents.
Answer:
a change of seasons. a volcanic eruption. phase changes of the Moon. high and low tides.
Explanation:
The amiodarone belongs to <u>class III </u>of the Vaughn-Williams classification system of antidysrhythmic medications.
<h3>
What is antidysrhythmic medications?</h3>
Antiarrhythmic drugs comprise many different drug classes and have several different mechanisms of action. Furthermore, some classes and even some specific drugs within a class are effective with only certain types of arrhythmias. Therefore, attempts have been made to classify the different antiarrhythmic drugs so by mechanism. Although different classification schemes have been proposed, the first scheme (Vaughan-Williams) is still the one that most physicians use when speaking of antiarrhythmic drugs.
The following shows the Vaughan-Williams classification and the basic mechanism of action associated with each class.
- I - Reduce phase 0 slope and peak of action potential.
- IA - Moderate reduction in phase 0 slope; increase APD; increase ERP.
- IB - Small reduction in phase 0 slope; reduce APD; decrease ERP.
- IC - Pronounced reduction in phase 0 slope; no effect on APD or ERP.
- II - Block sympathetic activity; reduce rate and conduction.
- III - Delay repolarization (phase 3) and thereby increase action potential duration and effective refractory period.
- IV - Block L-type calcium-channels; most effective at SA and AV nodes; reduce rate and conduction.
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