Answer:
Muscarine binding to a G protein-coupled receptor results in the activation of the amplifier enzyme adenylate cyclase, and formation of cAMP.
Explanation:
Because I thought the mechanism by which the muscarinic receptor inhibits adenylyl cyclase is through activation of an inhibitory GTP-binding protein, Gi. The α subunit of Gi competes with the α subunit of the G protein activated by stimulatory agonists (GS) for regulation of adenylyl cyclase.
I think!
Answer:
the answer is A
Explanation:
animal cells do not have cell walls or chloroplasts.
Given what we know, we can confirm that ready-to-eat TCS food must be date marked if it will be stored for longer than 24 hours.
<h3>What is TCS food?</h3>
- TCS food refers to food that must be stored in temperature-controlled environments to ensure safety.
- These foods, if not stored properly and at the correct temperatures can cause sickness to the consumers.
<h3>Why does it need to be date-marked?</h3>
- The reason the foods need to be date-marked is to relay information to the potential consumer as to by when the food should be consumed.
- This is done to ensure that if stored correctly, the food does not have time to spoil and cause health risks for consumers.
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Therefore, we can confirm that ready-to-eat TCS food must be date marked if it will be stored for longer than 24 hours in order to <em><u>protect the </u></em><em><u>health </u></em><em><u>of the </u></em><em><u>consumers</u></em><em><u>. </u></em>
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To learn more about TCS food visit:
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Answer: Today, almost all evolutionary relationships are inferred from molecular sequence data. This is because: DNA is the inherited material. We can now easily, quickly, inexpensively and reliably sequence genetic material.
Explanation:
Answer: Cystic Fibrosis
Explanation:
Cystic fibrosis (abbreviated CF) is an autosomal recessive genetic disease that mainly affects the lungs, and to a lesser extent the pancreas, liver and intestine, causing an abnormally thick, sticky mucus to build up in these areas. This mucus collects in the airways of the lungs and pancreas. The main cause of morbidity and mortality is pulmonary involvement, which accounts for 95% of deaths, mainly due to repeated infections caused by bronchial obstruction due to the secretion of very thick mucus.
This build up of mucus causes life-threatening lung infections and serious digestive problems. It is one of the most common types of chronic lung disease in children and young adults, and is a life-threatening disorder; patients often die from lung infections due to <em>Pseudomonas</em> or <em>Staphylococcus</em>.
<u>It is a hereditary disease produced by a mutation in the gene encoding the cystic fibrosis transmembrane conductance regulator (CFTR) protein. This protein is involved in the passage of chlorine ion through cell membranes and its deficiency alters the production of sweat, gastric juices and mucus. </u>The disease develops when neither allele is functional. Over 1500 mutations have been described for this disease, most of which are small deletions or point mutations; less than 1% are due to mutations in the promoter or chromosomal rearrangements. However, many people carry the CF gene, but do not have any symptoms. This is because a person with this disease must inherit 2 defective genes, 1 from each parent.
<u>There is no curative treatment, however there are treatments that allow the improvement of symptoms and extend life expectancy. In severe cases, the worsening of the disease may necessitate a lung transplant.</u>
