Answer:
Muscle contraction function.
Explanation:
The nerve endings possess synaptic acetylcholine vesicles ready to be released. The action potential depolarizes the presynaptic terminal and increases the concentration of axoplasmic calcium; Acetylcholine molecules are thus released, so that the concentration of the neurotransmitter at postsynaptic (nicotinic) receptors is temporarily increased. This is followed by post-synaptic membrane depolarization, muscle membrane action potential with increased rnioplasmic calcium concentration, and finally muscle contraction. Acetylcholine is hydrolyzed by acetylcholinesterase and resynaptic at the presynaptic level by cholinecetyltransferase. The etiopathogenesis of myasthenia gravis is autoimmune and there are antibodies against acetylcholine receptors that circulate in the blood, as well as a decrease in the number of receptors on the motor plates, that is, it is produced by the postsynaptic blockage of the myoneural plaque, that generates fatigue and localized or generalized muscle weakness that is characterized by the worsening of the contractile force of the muscle.
Answer:
Yesssss mercury Venus earth Mars Jupiter Saturn Uranus Neptune and my baby pluto
Answer: Deflation
Explanation: it’s correct
I believe it's called a Basalt.
Hope this helps..
Answer:
Muscular dystrophy
Explanation:
Muscular dystrophy is an inheritable genetic condition that involves mutations in genes that encodes the production of muscle proteins to build and to preserve healthy proteins. The disease is characterised by progressive weakness and loss of muscle mass.
Dystrophy is referred to as a disorder in which the a tissue or an orgasm progressively wastes away. Thé dystrophy term is used because the disease involves the gradual and progressive wasting away of the muscle which is a type of tissue causing muscular weakness allowing the child to experience these symptoms.
