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OlgaM077 [116]
3 years ago
9

If you were studying the causes of cancer, which topic might interest you?

Biology
2 answers:
nikdorinn [45]3 years ago
5 0

spindle-fiber structure because this is used to divide cells; cancer is just cells dividing way too much. so if the <em>spindle-fiber structure </em>were to fail it could cause cancer.

Mariana [72]3 years ago
5 0

Answer:

cyclin-dependent kinases

Explanation:

The cyclin-dependent kinases (CDKs) are critical proteins that control the progression in the cell cycle and pathways associated with cancer progression (for example, apoptosis). These proteins phosphorylate serine or threonine residues of target factors involved in gene transcription

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After the depletion of hepatic glycogen in newborns, which compounds can be used as precursors to sustain the blood glucose leve
VashaNatasha [74]

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Nutrients from breast milk.

Explanation:

Healthy newborns make blood glucose from sugar and several nutrients from the colostrum, a type of liquid that mother’s breasts produce before breast milk itself. Later, the babies make glucose from mature breast milk.

Most healthy babies, born after 37 weeks of gestation do not risk hepatic glycogen drops. They can easily compensate for normal drops in blood sugar, in other words, whenever the baby is breastfed when needed, he/she will be able to keep  his/her glucose levels stable.

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chimpanzees reproduce sexually and sponges reproduce asexually what disadvantage do chimpanzees have compared to sponges? A) chi
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Read 2 more answers
Two protein kinases, K1 and K2, function sequentially in an intracellular signaling pathway. If either kinase contains a mutatio
Korolek [52]

Answer:

The order must be K2→K1, since the permanently active K1 allele (K1a) is able to propagate the signal onward even when its upstream activator K2 is inactive (K2i). The reverse order would have resulted in a failure to signal (K1a→K2i), since the permanently active K1a kinase would be attempting to activate a dead K2i kinase.

Explanation:

  • You characterize a double mutant cell that contains K2 with type I mutation and K1 with type II mutation.
  • You observe that the response is seen even when no extracellular signal is provided.
  • In the normal pathway, i f K1 activat es K2, we expect t his combinat ion of two m utants to show no  response with or without ext racell ular signal. This is because no matt er how active K1 i s, it would be unable to  act ivate a mutant K2 that i s an activit y defi cient. If we reverse the order, K2 activating K1, the above  observati on is valid. Therefore, in the normal signaling pathway, K2 activates K1.
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3 years ago
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