Answer:
The correct answer will be:
1. Acetylcholine binds to receptors on the motor end plate
2. Chemically-regulated ion channels open, causing depolarization
3 End plate potentials trigger action potential(s).
4. Transverse tubules convey potentials into the interior of the cell
5. Ca++ is released from the sarcoplasmic reticulum
6. Ca++ binds to troponin, pulling on tropomyosin
7. Binding sites on actin are uncovered, allowing myosin to bind and carry out power strokes
8. Force increases.
9. Ca++ is pumped (re-sequestered) into the sarcoplasmic reticulum
10. Force decreases.
Explanation:
The muscle contraction is a highly controlled mechanism which begins at the neuromuscular junction with the release of the acetylcholine neurotransmitter. This neurotransmitter causes the depolarization of the membrane by binding to the receptors of the motor end plate which generates an action potential. This action potential is transmitted via T-tubules from sarcolemma to the sarcoplasmic reticulum.
The sarcoplasmic reticulum releases calcium ions which binds to the troponin protein. This troponin removes the protein tropomyosin from the actin causing the rotation of the tropomyosin exposing the binding sites for myosin. The myosin binds to the actin using energy from the ATP which pulls the actin causing contraction. Another ATP binds the myosin head which weakens the bond between myosin and actin which releases the myosin which decreases the force between them decreases and the muscles relax.
Because certian vitals can lead to different things and if the medical professional is about to make a diagnosis and there’s a certian vital that should not be there it can change the diagnosis they are about to make.
The nurse administers cimetidine to a 75-year-old client diagnosed with a Gastric ulcer the nurse should monitor the client for the following adverse reactions Headache, Sleepiness, Confusion, Dizziness, Nausea which contribute for the change in Metal status.
- Cimetidine is a H
Receptor Antagonist. - Gastric partial cells contains receptors for Histamine.
- Histamine is released from the Enterochromaffin cells upon the stimulation from food intake or from Vagus nerve stimulation.
- Thus Histamine released from Enterochromaffin cells binds to H receptors on the parietal cells and increases the secretion of HCl.
- Cimetidine has totally opposite effect and decreases the secretion of HCl from the parietal cells.
- Not only on gastric cells H receptors are also present in CNS where they stimulate brain cells.
- The above point justifies the adverse effects of Cimetidine.
- Patients over the age of 50 or those who are severely ill may experience transient confusion while taking H
blockers, particularly cimetidine.
Hence from these points we can conclude that patients who are above 50 years and receiving Cimetidine medication for gastric ulcer should be monitored for the above mentioned adverse reactions.
Learn more about Gastric Ulcer:
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Answer:
Explanation: amnesia is a condition that gets better over time so it's normal that his game gets better the more he plays. He will gradually get his memory back
