1. Each of three common form of galactosemia involved impaired utilization of glucose because certain enzymes which are very important to galactose metabolism are absent. The absence of these enzymes prevents the body from properly metabolism galactose, thus, the sugar is found in the blood at high concentration.
2.Galactosemia is a disorder which affects how the body processes a sugar called galactose. In this disorder, too much galactose is found in the blood. There are three major forms of galactosemia, these are:
1. Type 1 galactosemia {Classic galactosemia} is the most severe form of this disease. It is an inborn error of galactose metabolism, which is caused by the deficiency of the enzyme galactose 1 phosphate uridylyltransferase. Thus, the reaction catalyzed by this enzyme is blocked in this disorder.
2. Type 2 galactosemia is an autosomal recessive metabolic disorder, that is marked by accumulation of galactose and galactitol. The enzyme that is deficient in this disroder is galactokinase. The chemical reaction catalyzed by this enzyme is blocked in this disorder.
3. Type 3 galactosemia is a rare autosomal recessive form of galactosemia, which is associated with the deficiency of the enzyme galactose epimerase. Thus, in this disorder, the biochemical reaction, which is catalyzed by galactose epimerase is blocked.<span />
The most important idea is that the genetic material of any organism must be able to accurately replicate itself at least every generation (or for multicellular organisms at each cell division).
Base pairing (A-T or U and C-G)allows DNA and RNA (eg in polio virus, see Wikipedia page on RNA dependent RNA polymerase) to create a copy of themselves, when the appropriate enzymes are present. Proteins have no way of making a copy of themselves.
Stability is probably the main reason DNA is the most common genetic material. DNA has no enzymatic activity and was probably selected for to maintain the integrity of the genetic material (rather than having to perform a function for the cell/virus, during which it may be destroyed). The double helix structure also protects its integrity, and proofreading enzymes have also evolved which correct most of the mistakes made at DNA replication. RNA viruses don't have this mechanism- which could be said to be an advantage (as they can rapidly change and therefore avoid their hosts' immune systems), however in non-parasitic organisms most mutations in a gene would lead to a loss of an essential function and the extinction of that genome.
I don't think either of these reasons are relevant, but I think the main reasons retroviruses convert their RNA to DNA are so they can use the host cell's replication machinery (this was they do not need to encode as many genes), and secondly they need avoid the antiviral mechanisms of the cell, which would destroy any double stranded RNA molecules found (even if the virus was single stranded, dsRNA would have to be produced at replication).
Answer:
1/4
Explanation:
Given that the disease cystic fibrosis is caused by a recessive allele (f), the individuals with genotype "ff" will express the disease. Both "FF and Ff" genotype will give the individuals a normal phenotype.
Both the parents are carrier (Ff) for the disease. They have 3/4 probability of getting a healthy child and 1/4 probability of getting a child with cystic fibrosis.
I’m pretty sure that’s right
I dont want to mislead you, but I believe the answer is A because letter B is wrong because plants go through cellular respiration and photosynthesis, whereas animals go through cellular respiration but not photosynthesis, and for C, I've never heard of that so it doesnt seem likely.