Answer:
Glycine is required for purines, aspartate for pyrimidines, glycine and aspartate for both purines and pyrimidines. The remaining amino acids are not required for the synthesis.
Explanation:
Nucleotides are the monomer for the synthesis of DNA and RNA. The two main class of nitrogenous bases are purines and pyrimidines. These nitrogenous bases require the amino acids as a precursor for their synthesis.
Glycine is the simplest amino acid and required for the synthesis of purines. Aspartate is required for the synthesis of pyrimidines. Glutamine and aspartate are required for the synthesis of both purines and pyrimidines. Lysine, leucine, alanine, histidine, methionine, tryptophan and alanine are not used as a precursor for the nucleotides.
Answer:
I think is C.
Explanation:
Oh and the mouse is not cute.
The correct answer is phosphorylation. When an ADP (Adenine Diphosphate) molecule gains a phosphate, it becomes an (Adenine Triphosphate) molecule. This process is called a phosphorylation. Phosphorylation and its counterpart dephosphorylation change many proteins in terms of its functions and activities.
Answer:
B. The father did not contribute a sex chromosome to his daughter due to nondisjunction of the sex chromosomes. The daughter is XO and her only X chromosome came from her mother, who was a carrier.
A.The mother's X chromosomes failed to separate during meiosis, and the daughter inherited two X chromosomes with the Lesch-Nyhan mutation. The father contributed no sex chromosomes.
Explanation:
As seen in the question above, a little girl was diagnosed with Lesch-Nyhan syndrome, which is an X-linked recessive condition caused by a mutation in the HPRT1 gene responsible for purine metabolism.
The little girl's parents do not have the syndrome, and no one in the little girl's paternal family presented this syndrome, however, we know that the maternal grandfather of the little girl's mother had the syndrome, which means that it was the mother's genetic material that contributed to the development of the syndrome in the little girl. This was because the little girl did not receive any X chromosomes from her father, but she inherited the two X chromosomes from her mother that coded for the Lesch-Nyhan mutation. This happened because the mother's X chromosome disjuction did not occur during meiosis I.
As shown above, the father did not contribute any sex chromosomes to his daughter, which means that the daughter is XO and her only X chromosome came from her mother, who was a carrier.
