Hormone supplements and dietary supplements, and relaxation techniques
Ff- father
ff- mother
i am pretty sure just learned it like 3 yrs ago if not then its the opposite
Answer:
C. Electron beams have much shorter wavelengths than visible light
Explanation:
A beam of electrons is used to illuminate and create magnified images of specimens in an electron microscope. The resolution of a microscope increases with a decrease in the wavelength of the light it uses for illumination. The wavelength of electrons used in the electron microscope is about 100,000 times shorter than that of visible light. Therefore electron microscopes have a resolution thousand times better than the light microscope.
Answer: Osmosis
Explanation:
"Water makes up a large percentage of the body's cells. For a cell to remain in homeostasis, there must be a mechanism to control water changes in the cells. The movement of water from an area of high water concentration to an area of low water concentration is osmosis." Source, Quizlet. https://quizlet.com/350562884/cell-homeostasis-flash-cards/
Long-term potentiation (LTP) is considered a cellular correlate of learning and memory. The presence of G protein-activated inwardly rectifying K(+) (GIRK) channels near excitatory synapses on dendritic spines suggests their possible involvement in synaptic plasticity. However, whether activity-dependent regulation of channels affects excitatory synaptic plasticity is unknown. In a companion article we have reported activity-dependent regulation of GIRK channel density in cultured hippocampal neurons that requires activity oF receptors (NMDAR) and protein phosphatase-1 (PP1) and takes place within 15 min. In this study, we performed whole-cell recordings of cultured hippocampal neurons and found that NMDAR activation increases basal GIRK current and GIRK channel activation mediated by adenosine A(1) receptors, but not GABA(B) receptors. Given the similar involvement of NMDARs, adenosine receptors, and PP1 in depotentiation of LTP caused by low-frequency stimulation that immediately follows LTP-inducing high-frequency stimulation, we wondered whether NMDAR-induced increase in GIRK channel surface density and current may contribute to the molecular mechanisms underlying this specific depotentiation. Remarkably, GIRK2 null mutation or GIRK channel blockade abolishes depotentiation of LTP, demonstrating that GIRK channels are critical for depotentiation, one form of excitatory synaptic plasticity.
Learn more about receptors here:
brainly.com/question/11985070
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