A number of different types of mutations in the HBB gene can cause human β-thalassemia, a disease characterized by various level
s of anemia. Many of these mutations occur within introns or in upstream noncoding sequences. Explain why mutations in these regions often lead to severe disease, although they may not directly alter the coding regions of the gene.
Mutations in introns create alternative splicing site which in turn make mRNA with reduced or no translation.
Explanation:
Introns regulate the splicing of heterogenous RNA during post transcriptional modifications as well as affect the stability of mature mRNA. The stable mRNA is more likely to be translated into proteins. So, introns also regulate the translation of mRNA.
In human beta thalassemia, mutations in intron create alternative splicing sites which in turn affect the formation of beta globin chain of hemoglobin. Lack of beta globin chain reduces the amount of functional hemoglobin and causes anemia.
<h2><u><em>Upon reinfection, the memory cells will immediately differentiate into plasma cells and CTLs without input from APCs or TH cells.</em></u></h2>
Explanation:
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The answer is LH only.
Luteinising hormone promotes the release of an egg
from an ovary during ovulation. Follicle Stimulating Hormone, on the other hand, stimulates
the maturation of ovarian follicles. When its levels
drop and LH surge, ovulation of a mature
egg from the mature follicle occurs.
YES BECAUSE THE BOTTOM OF THE FOOD CHAIN IS THE PRODUCER AND THAT IS THE GRASS AND THE ORGANISM THAT EAT THE GRASS WOULD DIE BECAUSE THAT ORGANISM WOULDNT BE ABLE TO EAT AND THEN THE SAME THING WILL HAPPEN TO THE OTHERS.
