Answer:
B) a nonsense mutation; this is because a nonsense mutation results in the change of a regular amino acid codon into a stop codon, which ceases translation. This fits with the problem's description of the protein that causes the symptoms as too short, as translation is the process by which proteins/polypeptides are created. A missense mutation would not be the answer because it still codes for an amino acid, which would not shorten the protein. A duplication of the gene would probably just lengthen the protein or not affect its length at all.
<span>it is Cardiac muscle tissue. Cardiac muscle tissue is an extremely specialized form of muscle tissue that has evolved to pump blood throughout the body. In fact, cardiac muscle is only found in the heart and makes up the bulk of the heart’s mass. The heart beats powerfully and continuously throughout an entire lifetime without any rest, so cardiac muscle has evolved to have incredibly high contractile strength and endurance. And because the heart maintains its own rhythm, cardiac muscle has developed the ability to quickly spread electrochemical signals so that all of the cells in the heart can contract together as a team....</span>
I'll say that it is +1 since hydrogen is desperate to lose an electron to fulfill the octave rule (that the molecules want to gain 8 electrons and if they can't do it, they want to lose all their electron). Electron is - so if it lose a electron, it becomes +.
Hopefully this helps.
PS. I'm not certain of my answer but it is logical.
The zygosporangium of Rhizopus stolonifer function to
ensure the survival of the species by remaning dormat during unsuitable
conditions. I am hoping that this
answer has satisfied your query and it will be able to help you in your
endeavor, and if you would like, feel free to ask another question.
