Outer membrane or plasma membrane, cytoplasm, nucleus, ribosomes,
Answer:
Repair mechanism for base cleavage (BER)
Explanation:
Repair by base cleavage (BER)
The altered bases are specifically recognized by glycosylases and removed, generating an AP site. The hole is filled by a DNA polymerase that takes the healthy strand as a template. This system arises not only by exposure to external agents, but also by the cell's own activity.
In case of damage in more than one nucleotide, repair by nucleotide excision (NER) is performed.
Nucleotide excision repair (NER)
The damaged area is recognized by UvrA and B, then A and B separate and UvrC enters which forms a complex with endonuclease activity with B. This enzyme cuts the T-dimer and the gap is filled by a DNA polymerase. There is also the TC-NER system (transcription-coupled nucleotide repair system). The alteration of these mechanisms gives rise to diseases such as: Xeroderma pigmentosum, Trichotiodystrophy or Cockayne Syndrome
Predators are the ones who consume the prey and rely on them as a food source, while prey rely on predators to control their population.
Answer:
systemic lupus erythematosus
Explanation:
Systemic lupus erythematosus (SLE or just lupus) is a chronic inflammatory disease of autoimmune origin, the symptoms of which may appear in various organs slowly and progressively (in months) or more rapidly (in weeks) and vary with phases of activity and remission. Because it is a disease of the immune system, which is responsible for producing antibodies and organizing inflammation mechanisms in all organs, when a person has SLE they may have different types symptoms and various body locations. Some symptoms are general such as fever, weight loss, loss of appetite, weakness and discouragement. Others, specific to each organ such as joint pain, skin blemishes, pleural inflammation, hypertension, and / or kidney problems.
The main cause of this disease is the imbalance in the production of antibodies that react with proteins in the body itself and cause inflammation in various organs such as the skin, mucous membranes, pleura and lungs, joints, kidneys, etc.). Thus, we understand that the type of symptom a person develops depends on the type of autoantibody the person has, and that as the development of each antibody relates to the genetic characteristics of each person, each person with lupus tends to have clinical manifestations. specific and very personal (symptoms).
Autoantibodies are antibodies directed to the body's own cells and tissues. Normally, the immune system differentiates the body's own proteins from foreign proteins, forming antibodies only against those identified as potentially dangerous.
Explanation:
Genetic variation is increased by meiosis
Because of recombination and independent assortment in meiosis, each gamete contains a different set of DNA. This produces a unique combination of genes in the resulting zygote. Recombination or crossing over occurs during prophase I
