When a DNA sequence alteration results in a stop codon rather than a codon that specifies an amino acid, it is known as a nonsense mutation. This is rarely seen in 10% of patients with genetic disease.
<h3>What is nonsense mutation?</h3>
A nonsense mutation in a DNA sequence causes a premature stop codon, also known as a nonsense codon, in the transcribed mRNA as well as a shortened, ineffective, and typically nonfunctional protein product.
Because stop codons, also known as nonsense codons, signal the completion of protein synthesis rather than encoding for an amino acid, they are the source of the term "nonsense mutation."
Examples of illnesses for which nonsense mutations have been implicated as contributing factors include: Cystic fibrosis (produced by the G542X mutation in the cystic fibrosis transmembrane conductance regulator); (CFTR) Beta-globin (thalassemia) Hurler disease.
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One reason is if someone is injured and needed to use pain pills, sometimes they get addicted to it
Answer: B (Color blindness)
Explanation:
Typical red-green color blindness in human patients is caused by mutations on genes located in the X chromosome. These mutations act in a recessive manner. Since females have two X chromosomes, the presence of a mutation in a single one of them does not normally result in color blindness. Males, in contrast, have a single X chromosome and therefore the presence of a mutation is likely to cause the disease.
About the other options: Down‘s syndrome is a numerical chromosomal anomaly, not related to sex. Human blood type is a codominant trait. Finally, tail length in dogs is a polygenic trait not amenable to classic Mendelian analysis.
Answer:
D. decreased supply of energy
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Answer:
ETS is responsible for more lung cancer deaths than heart disease deaths annually
