Answer:
The correct option is b. DNA ligase
Explanation:
DNA ligase is an enzyme that joins DNA fragments. If two DNA fragments or molecules have complementary ends, the ligase can join them together to form a single and intact DNA molecule, without interruptions. This is because the ligase enzyme seals the space between the molecules to form a single DNA fragment.
The others enzymes have different functions:
DNA helicase is characterized by separating DNA from double strands into single strands and acts in DNA replication.
ATP methylase plays an important role in DNA replication when determining the beginning of replication that has already been replicated or not.
DNA polymerase is an enzyme that is responsible for catalyzing the polymerization of the new strand of DNA during the replication of this molecule.
Long-term potentiation (LTP) is considered a cellular correlate of learning and memory. The presence of G protein-activated inwardly rectifying K(+) (GIRK) channels near excitatory synapses on dendritic spines suggests their possible involvement in synaptic plasticity. However, whether activity-dependent regulation of channels affects excitatory synaptic plasticity is unknown. In a companion article we have reported activity-dependent regulation of GIRK channel density in cultured hippocampal neurons that requires activity oF receptors (NMDAR) and protein phosphatase-1 (PP1) and takes place within 15 min. In this study, we performed whole-cell recordings of cultured hippocampal neurons and found that NMDAR activation increases basal GIRK current and GIRK channel activation mediated by adenosine A(1) receptors, but not GABA(B) receptors. Given the similar involvement of NMDARs, adenosine receptors, and PP1 in depotentiation of LTP caused by low-frequency stimulation that immediately follows LTP-inducing high-frequency stimulation, we wondered whether NMDAR-induced increase in GIRK channel surface density and current may contribute to the molecular mechanisms underlying this specific depotentiation. Remarkably, GIRK2 null mutation or GIRK channel blockade abolishes depotentiation of LTP, demonstrating that GIRK channels are critical for depotentiation, one form of excitatory synaptic plasticity.
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Answer:
The answer to this question is D. They multiply rapidly to increase their population. I sure do hope this helped dear! XD
Explanation:
Explanation:
The answer is mitochondria because this is the super producer of cell systems. while an active animal, (squirrel, rodents, horses, deer etc.) are all prey items for preditors, they possess more energy to survive & escape danger...