Some bacteria are not able to function properly and replicate at the fever-high temperatures hence this reduces their infection rates in the body. More research has also shown that the temporal increase in body temperature, during the fever, increases the activity of the cytotoxic T-cells that are significant in fighting infection.
<h2><em>the DNA that is packaged into chromosomes must be fully copied. Previous to replication, a germ cell contains two copies of each chromosome, a maternal copy, and a paternal copy. ... DNA replication occurs in the same fashion as it does during mitosis.</em></h2>
Having blood glucose levels that are too high or too low is very dangerous. Having a blood glucose level that is too high results in high blood pressure. Having a low blood glucose level causes hypoglycemia. Hypoglycemia causes symptoms like dizziness, fatigue, exessive sweating, and shaking.
B
<u>Temperature</u> is NOT a density-dependent factor affecting population growth
Explanation:
Density-dependent factors mean variables that are affected by the density of the population. The opposite of density-depend is density-independent factors. These are those variables that affect the density of the population but are not affected by the density of the population.
In this case, temperatures are not affected by the density of the population. However, temperatures affect the density of the population. Most individuals can only survive within an optimal temperature range.
The rest of the choices are density-dependent variables
Learn More:
For more on dependent and independent variables check out;
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Answer:
D) observation that certain tumor-suppressor genes and oncogenes are involved in a sequential manner in the development of colon cancer
Explanation:
Cancer is defined as the abnormal growth of cells of the body. These cells multiply/replicate without control thereby forming a lump or tumor.
Cancer growth has 5 stages and they go though different multi level steps.Different finding backs this up and it includes observation about certain tumor-suppressor genes and oncogenes being involved in a sequential manner in the development of colon cancer.