Achondroplasia is a type of dwarfism in which the long bones stop growing during childhood, resulting in limbs that are dispropo
rtionately shorter than the torso. This genetic disorder is characterized by deficiencies in the epiphyseal plate that include a low number of chondrocytes and inability of chondrocytes to enlarge. Which zones do you think would be most affected by this disorder? Achondroplasia is a type of dwarfism in which the long bones stop growing during childhood, resulting in limbs that are disproportionately shorter than the torso.
This genetic disorder is characterized by deficiencies in the epiphyseal plate that include a low number of chondrocytes and inability of chondrocytes to enlarge.
Which zones do you think would be most affected by this disorder?
a. Hypertrophic and Calcification
b. Zones Hypertrophic and Resting
c. Zones Proliferation and Hypertrophic
d. Zones Proliferation and Calcification Zones
The best answer to the question: Which zones do you think would be most affected by this disorder, would be, C: Proliferation and hypertrophic zones.
Explanation:
The reason why a human being is able to grow from his/her small body when borh, to his/her adult body size is in part because his/her bones continue developing and growing, meaning enlarging, until they reach their adult, and fully matured size. However, in people with Achondroplasia this is not the case. Essentially, people with this affection will have problems at the zones of the bone where both proliferation of the chondrocytes and hypertrophy of the tissue that has been produced takes place. This will cause for an incomplete enlargement and formation of the fully-grown and development of the bone, and which becomes evident in people with this affectation due to their limbs being more "child-like" than in adults who did not suffer from it.
Cephalosporins (are bactericidal and have the same mode of action as other β-lactam antibiotics (such as penicillins) but are less susceptible to β-lactamases.) Cephalosporins disrupt the synthesis of the peptidoglycan layer forming the bacterial cell wall