Answer:
(a) The rate of oxygen consumption will decrease.
Explanation:
The rate of oxygen consumption will decrease due to the Potassium cyanide which inhibits both utilization of oxygen by the cell as well as ATP production. Cytochrome performs an important function in the mitochondria of the cell which is the production of ATP. Cyanide binds to the cytochrome and inhibits the ATP production that is performed by mitochondria of the cell. So Potassium cyanide decreases the rate of oxygen consumption in mitochondria.
Systematic observation, measurements and experiment and the formulation .testing.and modification of hypothesis
Answer:
If you ate the cake the protein in the cake would become denatured
Explanation:
As you eat the cake the salivary amylase in the mouth will begin to digest the carbohydrates. in the stomach the pH is around 2. This is a highly acidic environment.
At this point in digestion the stomach acids present will begin to act on the protein inside the cake. This protein will become denatured firstly, because of the stomach environment.
The cake will continue to pass along the digestive tract hereafter.
A solution with a pH of 7.0 is considered to be neutral
Answer:
The correct answer is ''All three are caused by disparate genes, but each gene was found to contain repeats of a unique trinucleotide sequence.''
Explanation:
Four groups of trinucleotide repeats related to DNA expansion have been characterized in the human genome. CGGI GCC, CAGIGTC, CTGIGAC and recently GW CTT sequences. The CGGl GCC sequences are usually located in the noncoding regions of different genes and when expanded they associate with fragile sites on the chromosomes. The CAGIGTC repeats are part of the coding region and are related to different neurodegenerative disorders. You can try to make a classification depending on the unit that is repeated, having on one side a group that are caused by repeats of the CAG trinucleotide (cytosine, adenine, guanine). Huntington's disease (HD) is a progressive disorder of motor, cognitive and psychic function. The disease is inherited in an autosomal dominant manner, and is caused by the expansion of CAG trinucleotide repeats in exon 1 of the gene called huntingtin (HTT or IT-15). Unstable mutations have also been found in which the repeat unit does not is composed of three nucleotides, if not four, such as type 2 myotonic dystrophy, where the tetranucleotide CCTG repeats. Fragile X Syndrome consists of an abnormal expansion of the cytosine-guanine-guanine trinucleotide (CGG) in the FMR1 gene, which prevents intellectual disability and also influences the formation of connective tissue