<span>If vinblastine prevents assembly of the spindle, then it interrupts cell division in Profase 1, when the assembly of the spindle begins. Paclitaxel prevents the depolymeritation of the spindle, so it will interrupt cell cycle in Anaphasis, when the dissasembly of spindle takes place.</span>
Answer:
The most likely explanation for the truncated polypeptide -due to the substitution of cytosine for adenine- is that mutation introduced a stop codon in the middle of the gene.
Explanation:
A codon consists of three nucleotides -in the RNA chain- whose order determines a specific amino acid. Not all codons code for amino acids, as there are termination codons, also called stop codons, which are UAG, UGA and UAA.
If in Manny's computer model, the substitution of cytosine by adenine produced a termination codon, the synthesis of a peptide is stopped prematurely resulting in a truncated peptide.
Learn more:
Stop codon brainly.com/question/6183177
The fructose 1-phosphate pathway can deplete intracellular phosphate/ ATP.
Explanation:
Fructose 1-phosphate is a derivative of fructose. For understanding in better way fructose metabolism has three enzymes. Fructose- bisphosphate aldolase B, fructokinase and Adenosine triphosphate. These all are present in liver and kidney of human as well rat. In liver rapidly fructose is change to fructose 1 through fructokinase.
After it is converted into trioses dihydroxyacetone phosphate as well as glyceraldehyde through aldolase. With glucose metabolism Fructose get synergistic effect
2 Homologous chromosomes in meiosis, and then replicate into sister chromatids.
Hope this helps, ( or is clear enough ) if not, comment below please!!!!
<span>the high expense; electron microscopes are costly</span>
