The answer would be, Heterozygous Genotype.
Hope that helps!!!!
Prader-Willi syndrome (PWS) is a gentic disorder which has an impact on numerous physiological systems. PWS affected individuals (specifically babies) experience delayed growth, significant hypotonia (low muscle tone), and feeding issues. It does effect circadian rhythms in mice models.
SNORD116, often referred to as HBII-85, is a non-coding RNA (ncRNA) molecule that contributes to the alteration of other small nuclear RNAs (snRNAs). Unlike the majority of other snoRNAs, SNORD116 is not significantly complementary to ribosomal RNA and is expressed widely in the brain (but not in PWS patients).
According to the studies, SNORD116 cause sleep defect in patients with Prader-Willi syndrome. Same observation was seen in mouse models too. Paternal expression of SNORD116 is thought to be a potential gene for the sleep disruptions/circadian rhythm’s that the majority of PWS sufferers.
To learn more about circadian rhythm click here
brainly.com/question/6434404
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Living things differences are important because it helps classify and signify one from another
They have a back bone
They are cold-blooded
Breathes through skin
(extras)
they go though metamorphosis
and they hatch from eggs! <span />
Answer:
Thyroid hormones
Explanation:
Animals and people exposed to pesticides such as insecticides, fungicides, and herbicides, have shown to be affected by their effect on the thyroid. These chemicals can produce endocrine alterations acting as thyroid disrupters. They affect many mechanisms in the organisms such as inhibition of thyroid iodine uptake, <u><em>interference with the thyroid hormone receptor</em></u>, binding to transport proteins, among others. They cause toxicity in the functioning of the hypothalamic-pituitary-thyroid axis.
There are many studies that associate thyroid diseases with exposure to organochloride pesticides. Significant alteration in the TSH, T3, and T4 levels have been expressed by people directly or indirectly exposed to the chemicals.
