Answer:
Hence, during infection in vivo, a noncytopathic virus may turn off the "differentiation" or "luxury" function of a cell while not killing that cell (loss of vital function). This is turn can disrupt homeostasis and cause disease
hope it helps you
Answer:
b. Frank received the mutant chromosome from his father. Nondisjunction occurred in his father during the first meiotic division.
Explanation:
As you can see in the question above, Frank has Klinefelter syndrome which causes him to have normal skin patches and skin patches without sweat glands. Her mother has completely normal hair, which may indicate that the defective gene was not supplied by her. In addition, Frank's father has anhydrotic ectodermal dysplasia, an X-linked condition where the skin does not contain sweat glands.
Although Frank's father's defective gene is linked to the X chromosome, it is likely that Frank inherited the defective gene from his country. This may have occurred because during meiosis I, his father's genes did not show disjunction. As a result, Frank presents a mosaic of his phenotype, because an inactivation of the X chromosome occurred.
The appropriate answer is D ! in this the dominant allele is not fully dominant ! this is seen in Snapdragon !
so answer is D
It's also known as ulnar drift.
Well...This might helps <span>Parallel venation is characteristic of monocots, the veins are usually parallel to each other along the length of the leaf. </span>
Pinnate venation is the veins are in a branching pattern, characterized by one major vein(called midrib) with smaller veins extending outward from it.
<span>Palmate venation is characterized by two or more major veins extending outward from one point like the fingers extending form the palm of a hand</span>
