During normal breathing, the brain is stimulated to breath with increasing acidity as a result of CO2 concentration from basic metabolic processes. The brain is quite selfish and only really wants to maintain it's pH which should be at a range of 7.3-7.45, and will not tolerate any decrease.
In patients who have a chronic respiratory disorder with things like COPD. The brain has become accustomed to excessive acidic content, and is now stimulated by the Hypoxic drive or by low oxygen content.
Thickening of gill epithelia in rainbow trout, caused by chloride cell proliferation, could lead to an impairment of oxygen uptake under moderate to severe hypoxia (Thomas et al 1988; Bindon et al., 1994; Greco et al., 1995).
<h3>What results in an increase in AMS in interstitial lung disease?</h3>
The number of alveolar macrophages (AMs) can rise in interstitial lung disease. Precursor cells from the peripheral circulation may have been drawn in, and/or there may have been local lung growth, to create this.
<h3>What connection does sarcoidosis have between lymphocytes and proliferating cells?</h3>
Additionally, a strong association between the quantities of lymphocytes and proliferative cells in sarcoidosis and fibrosis was discovered in bronchoalveolar lavage (BAL). Eosinophil counts and proliferating cell counts were positively associated in fibrosis.
<h3>How do AMS patients and healthy controls differ in terms of propagating AMS?</h3>
With a substantial association between these two indices, there was a considerable increase in proliferating AMs in all patient groups when compared to healthy controls (4.2 versus 1.4% Feulgen, and 2.1 against 0.5% Ki67).
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It slowly gets compressed so I guess you can say it moves slowly as the space gets compressed
The initial stage of atherosclerosis is characterized by accumulation beneath the endothelium of excessive amounts of LDL.
- The accumulation of lipids, cholesterol, and other chemicals in and on the artery walls is known as atherosclerosis. The accumulation is known as plaque.
- The build-up of lipids and fibrous components in the major arteries is a hallmark of the degenerative illness known as atherosclerosis. A picture of a typical artery's anatomy can be found in Foam cells, or subendothelial accumulations of cholesterol-engorged macrophages, are the earliest lesions of atherosclerosis.
- Nitric oxide and prostacyclin are two chemicals that are produced by your endothelium. These maintain the fluid in your blood and stop it from clotting when it shouldn't. These compounds' insufficient production is a symptom of various diseases, which raises your risk of clotting.
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The cell would have 2 nuclei but the cytoplasm wouldn't be able to split.
