Answer: option B) Secretin
Explanation:
Secretin is an enzyme formed by the upper intestinal layer and gets transported by blood to the PANCREAS which it stimulates: causing the flow of pancreatic juice, also the flow of bile and intestinal juice but to a lesser extent.
Alongside Gastrin, Secretin is one of the hormones of the Gastrointestinal tract; but their functions differ
Therefore, Secretin is the answer
Answer:Connective tissue integrates the various parts of the body and provides support and protection to organs. Muscle tissue allows the body to move. ... Different types of tissues form membranes that enclose organs, provide a friction-free interaction between organs, and keep organs together.
Explanation:
Answer:
<h2>Recombination rate or recombination frequency between locus A and locus C is either 2% or 50%.
</h2>
Explanation:
As given,
Recombination frequency between locus A and B is 35%, means that the distance between A and B is 35 cM.
Recombination frequency between locus B and C is 33% , means the distance between B and C is 33 cM.
Therefore, if the locus C is present between A and B, then the recombination frequency between A and C would be 2%
A-----C ----------B
A---C= 2%
A----B= 35%
B---C= 33%
So, here the recombination frequency between A and C is 2%, means their distance is 2 cM.
If the locus C is as, A---35---B--33----C ; A-----B------C
A-----B= 35%
B------C= 33%
A-----C= 33+35= 68%
As rule, maximum frequency can not exceed 50%, as here the Recombination frequency between A and C = 50%.
Answer:
the answer is A. E. coli B
Explanation:
The multiplicity of infection (MOI) refers to the ratio between the numbers of viruses used to infect <em>E. coli</em> cells and the numbers of these <em>E. coli </em>cells. Benzer carried out several experiments in order to define the gene in regard to function. Benzer observed that <em>E. coli </em>strains with point mutations could be classified into two (2) complementary classes regarding coinfection using the restrictive strain as the host. With regard to his experiments, Benzer observed that rII1 and rII2 mutants (rapid lysis mutants) are complementary when they produce progeny after coinfect E. coli K (where neither mutant can lyse the host by itself). The rII group of mutants studied by Benzer does not produce plaques on <em>E. coli</em> K strains that carry phage λ (lysogenic for λ), but they produce plaques on <em>E. coli</em> B strains. This study showed that rIIA and rIIB are different genes and/or cistrons in the rII region.
