This statement is false.
The human skeleton is the inner system of the body. It is made out of 270 bones during childbirth – this aggregate abatements to 206 bones by adulthood after a few bones have intertwined. The bone mass in the skeleton achieves most extreme thickness around age 30.
While looking at the figure, the union of the left and right brachiocephalic veins is Point A.
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What are brachiocephalic veins?</h3>
- The internal jugular vein and the subclavian vein join to form the brachiocephalic veins, also known as the innominate veins, which are substantial venous systems in the thorax.
- On the right side of the upper chest, the superior vena cava is formed by the union of the left and right brachiocephalic veins.
- These blood arteries are an essential part of the circulatory system in humans because they facilitate the removal of deoxygenated blood from the head and upper limbs.
- The internal jugular vein on the right and subclavian vein on the left combine to form the brachiocephalic veins. Both the vena cava and the brachiocephalic veins lack valves.
To learn more about brachiocephalic veins with the given link
brainly.com/question/28095282
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Question: Is the pre-synaptic or the post-synaptic neuron damaged? Explain your rationale in detail (in other words, detail how you came to your conclusion). You're going to have to really think on this one. It's not something you can look up in the text. You will need to use what you have learned about the autonomic nervous system and some critical thinking to arrive at the answer.
Horner's Syndrome riddle: Ach is the neurotransmitter at all ANS preganglionic fibers, including SNS. The pt has decreased sweating and pupil constriction, indicating blockage of SNS transmission. When the MD gave ACh it induced sweating, but not pupil dilation. This is because the sweat glands are an exception in normal SNS transmission, as the postganglionic fibers utilize ACh as their neurotransmitters. Other SNS pathways utilize NE as their neurotransmitter. So ACh acted directly on the sweat gland to produce sweating, but could not stimulate the typical postsynaptic neurons to release NE. When the MD gave NE he saw pupil dilation, but not sweating, indicating the NE worked directly at the iris to produce pupil dilation. This indicaates the problem was with the postsganglionic neuron. If the preganglionic neuron were the problem injecting ACh would have solved both problems.
Insulin and glucagon in glucose metabolism
