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nadya68 [22]
3 years ago
8

The first stage in the general adaptation syndrome is the alarm stage. * O True O False

Health
2 answers:
gladu [14]3 years ago
7 0
Answer:

true

explanation: t
serg [7]3 years ago
4 0

Answer:

True

Explanation:t

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Preservatives are added to foods to help prevent or slow spoilage due to bacteria, molds, fungi or yeast, as well as to slow or prevent changes in the food's color, flavor or texture, delay rancidity, and maintain freshness.

<em>                  Hope this helped! <3</em>

5 0
3 years ago
Read 2 more answers
If 2^x=3^y=12^z, show that 1÷z=1÷y+2÷x.....​
Ostrovityanka [42]

Answer:

Proof of \dfrac{1}{z}}=\dfrac{1}{y}+\dfrac{2}{x}  is shown below.

Explanation:

The given equation is

2^x=3^y=12^z

To prove : \dfrac{1}{z}=\dfrac{1}{y}+\dfrac{2}{x}

Proof : Let as assume,

2^x=3^y=12^z=k

2^x=k\Rightarrow 2=k^{\frac{1}{x}}    ...(1)

3^y=k\Rightarrow 3=k^{\frac{1}{y}}      ...(2)

12^z=k\Rightarrow 12=k^{\frac{1}{z}}    ...(3)

We know that

12=2\times 2\times 3

12=(2)^2\times 3

Using (1), (2) and (3), we get

k^{\frac{1}{z}}=(k^{\frac{1}{x}})^2\times k^{\frac{1}{y}}

k^{\frac{1}{z}}=k^{\frac{2}{x}}\times k^{\frac{1}{y}}

k^{\frac{1}{z}}=k^{\frac{2}{x}+\frac{1}{y}}

On comparing both sides, we get

\dfrac{1}{z}}=\dfrac{2}{x}+\dfrac{1}{y}

\dfrac{1}{z}}=\dfrac{1}{y}+\dfrac{2}{x}

Hence proved.

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3 years ago
compose a dialogue between two friend discussing about the academic environment of college and their preparation for exam.​
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2 years ago
My office need to deliver products to various sites in your town you can hire a firm to
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contracting is the correct answer!

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A new drug is developed that prevents the demyelinization occurring in the progress of multiple sclerosis. The drug protects the
Aleonysh [2.5K]

Answer:

Multiple sclerosis (MS) is the most common demyelinating and an autoimmune disease of the central nervous system characterized by immune-mediated myelin and axonal damage, and chronic axonal loss attributable to the absence of myelin sheaths. T cell subsets (Th1, Th2, Th17, CD8+, NKT, CD4+CD25+ T regulatory cells) and B cells are involved in this disorder, thus new MS therapies seek damage prevention by resetting multiple components of the immune system. The currently approved therapies are immunoregulatory and reduce the number and rate of lesion formation but are only partially effective. This review summarizes current understanding of the processes at issue: myelination, demyelination and remyelination—with emphasis upon myelin composition/architecture and oligodendrocyte maturation and differentiation. The translational options target oligodendrocyte protection and myelin repair in animal models and assess their relevance in human. Remyelination may be enhanced by signals that promote myelin formation and repair. The crucial question of why remyelination fails is approached is several ways by examining the role in remyelination of available MS medications and avenues being actively pursued to promote remyelination including: (i) cytokine-based immune-intervention (targeting calpain inhibition), (ii) antigen-based immunomodulation (targeting glycolipid-reactive iNKT cells and sphingoid mediated inflammation) and (iii) recombinant monoclonal antibodies-induced remyelination.Keywords: calpain, central nervous system, demyelination, fingolimod, glycolipids, lipids, multiple sclerosis, myelin, myelination, NKT cells, oligodendrocytes, remyelination, T cells

Explanation:

5 0
2 years ago
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