Answer:
The symptoms involved in ontogenesis imperfect are- bone breakage, pain in bone, deformation of bones, brittle teeth, coloured tint in the white portion of eyes, loss in hearing and loose joints.
Explanation:
In the disease, ontogenesis imperfect, the strong bone formation in body is prevented by the defective gene that produces collagen protein. People who have the deficiency of collagen by birth, can develop this disease or can have it by birth. This defect can be hereditary.
The symptoms included in this disease are the- deformation and breakage of bones, pain in bone, grey or purple tint in the white portion of the eye, short stature, brittle teeth, hearing impairment and loose joints.
Answer:
1
Explanation:
Stasis of milk in the mammary ducts, that's a common after delivery
<h2>O facial expressions</h2><h3>When you cry, you show emotions through your face. you are crying, and you're sad. Facial expressions show motions of the face that indicate feelings and emotions. So, the answer is facial expression.</h3>
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Evidence suggests that cAMP response-element-binding protein (CREB) may be a final common target of antidepressant treatment.
Antidepressant medications work by binding to a number of monoamine neurotransmitter receptors and activating the cAMP signal transduction pathway. Chronic antidepressant therapy has been linked to the cAMP signalling system. Increases in cAMP cause the cAMP response element-binding protein(CREB) to be phosphorylated more frequently via increased receptor adenylate cyclase activity .
CREB mediates transcriptional responses to increased cAMP levels by attaching to cAMP response element (CRE) sites.In order to prevent recurring episodes, treatment for depression should maintain therapeutic efficacy while also addressing behavioural symptoms and indicators of the illness. One important modulator of the transcriptional effects of antidepressants may be CREB.
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