Answer:
d. all endothelial cells would be venous
Explanation:
The Notch signaling pathway is a cell signaling system which consist basically of 4 NOTCH genes ( NOTCH1, NOTCH2, NOTCH3, and NOTCH4) each of them having the information to build a particular cell membrane receptor protein.
It has been demonstrated in zebrafish embryos that Notch signaling are highly determinant factors during vascular development, and a key function of these genes was specifically to regulate the differentiation of arterial fate in endothelial cells.
Notch signaling-deficient embryos showed a loss of expression of arterial markers such as ephrinB2 from arterial vessels with an accompanying expansion of venous markers into normally arterial domains. In contrast, embryos in which Notch signaling had been ectopically activated, meaning an activation out of "proper" place (where not expected, e.g. venous endothelial), exhibited the contrary phenotype, this is a suppression of vein-specific markers with ectopic expression of arterial markers in venous vessels.
To summarize, when Notch is supressed (in this case inhibited with a specific Notch inhibitor) arterial fate of endothelium is not followed, and venous fate is stablished. Thus, endothelial cells (those expected to form venous tissues but also arterial ones) would specified as venous.
Prokaryotes are organisms made up of cells that lack a cell nucleus or any membrane-encased organelles. Eukaryotes are organisms made up of cells that possess a membrane-bound nucleus that holds genetic material as well as membrane-bound organelles.
Answer:
parents with AB and O blood types can either have children with blood type A or blood type B. These two types are definitely different than parents' blood types!
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In diseases, similar patterns of mutations in harmless genes may possibly be a cause or an effect of the disease. To investigate if it is a cause, it is worth looking into the proteins synthesized by the gene and whether it’s structure or functionality is affected by the pattern observed. It is also worth looking into the downstream effects possibly caused by the pattern in the gene. The gene may encode a non coding region which could affect post transcriptional splicing for example.
<span>58/110 = . 53 ( >. 33 so it is an animal)</span>
