Urinary strictures
Bethanechol chloride (Urecholine) can be harmful to patients who have a urinary tract obstruction or bladder wall weakness. The medication has the ability to contract the bladder, causing pressure to build up in the urinary tract. In these patients, an increase in urinary tract pressure could cause the bladder to rupture.
What is Urinary retention?
- The inability to completely empty the bladder is referred to as urinary retention. The onset can be abrupt or gradual.
- Symptoms of sudden onset include inability to urinate and lower abdominal pain.
- When symptoms appear gradually, they may include loss of bladder control, mild lower abdominal pain, and a weak urine stream.
- If there is an obstruction or stricture (narrowing) in or around the bladder, or if the muscles in or around the bladder are weak, urine may be retained. Urinary retention can also be caused by certain types and locations of tumors, certain medications, being dehydrated, or having constipation.
To learn more about urinary retention from the given link
brainly.com/question/20681879
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Answer:
DO support the person's breathing by administering oxygen or performing rescue breathing. DO administer naloxone. DO put the person in the “recovery position” on the side, if he or she is breathing independently. DO stay with the person and keep him/her warm.
Explanation:
Answer:
a. Acetyl CoA
Explanation:
Acetyl-CoA is produced in the mitochondria by the process of oxidative decarboxylation of pyruvate and β-oxidation of the fatty acids.
However, <u>both acetyl CoA and Oxaloacetate cannot cross mitochondrial membrane.</u> But the two join together to form citrate and thus, citrate can cross the mitochondrial membrane.
Answer:
D. ADH
Explanation:
ADH also known as the antidiureitc hormone is secretated in response to low production. The angiotensin II (produced in the lungs) increase the volumen os ADH. As its name says this hormone avoids the water losses in the kidneys in order to do that ADH associates with the AVPR1A recpetors, those receptors open calcium compartiments inside the cells, once opened the vessel constrictions starts avoiding the water losses. Those receptos also stimulates platelet aggregation and coagulation factor VIII, by this way the ADH also avoid hemorrhages.