Answer: 1/4
Explanation:
Firstly, in order for both parents to be type A and have children with type o blood, their blood types must both be Ao. Since o is a recessive blood type, a punnett square shows there is a 25% chance any child of theirs will have type o blood. If neither parent is color blind and they have a son who is, it implies that the mother is a carrier of colorblindness and has the genotype XᴮXᵇ. If you do a punnett square of the not colorblind father (XᴮY) and the mother, it shows that a daughter would have a 0% chance of being colorblind. Therefore colorblindess is irrelevent, since there is no possibility of the daughter not having normal color vision. In conclusion, there's a 25% chance she will have type o blood and not be colorblind, since the other 75% chance would be having type A blood and not being colorblind.
Answer:
Restriction endonucleases are utilized to cut the deoxyribonucleic acid at a specific Restriction site. HindIII is an example of Restriction endonuclease. The Restriction site of HindIII is as per the following:
5'A*AGCTT3'
3'TTCGA*A5' (* represents restriction site)
The Restriction endonuclease cuts the particular gene from the foreign DNA and produces numerous fragments with the sticky ends. The part of the outside DNA which contains the gene of intrigue or a particular gene is ligated with the plasmid.
On the off chance that foreign DNA and plasmid DNA are separated by the same Restriction endonuclease, they will create sticky ends integral to one another. In this way, the segment of foreign DNA can be ligated to the plasmid DNA by the assistance of DNA ligase at the integral sticky end to shape a recombinant DNA.
Additionally, by the assistance of the same Restriction endonuclease for example HindIII for this situation, the recombinant plasmid can be treated with a similar Restriction endonuclease to extract the ideal gene or gene of intrigue.
The wound made from the cut, when only epidermis is damaged, is called an abrasion. Deeper wound injury, when dermis (deeper layer of the skin) is damaged is called laceration. An avulsion is more traumatic abrasion that removes all layers of skin.
Answer:
The answer to the question: What effect will the decreased iron availability have on the production of diphteria toxin, would be, A: The lack of co-repressor binding to DtxR will lead to a loss of repression and an increase in the production of toxin.
Explanation:
Diphteria is a very serious disease that is caused by the gram-negative bacteria Corynebacterium Diphtheriae, which attacks the upper respiratory tract, especially the throat, and causes a thick lining to form, complicating the normal respiratory function. In order to accomplish such a feat, this bacteria produces a toxin that attacks human cells. But the toxin will not be produced if there is a high availability of iron in the area where the bacteria intends to infect, because if that is the case, then iron will work as a co-repressor with DtxR to prevent the expression of the bacterial gene that codes for the toxin. A co-repressor means that iron will work as a coadjuvant for DtxR and will not allow the genes of the bacteria to produce the toxin. In the case of this question, the answer is A because we have a lack of iron availability, which means that DtxR will not be enough to stop the toxin from being produced.
An axon sends impulses away from the cell body. :)