<span>A drug used to treat CML, imatinib, binds to the active site of Abl kinase. Why does this drug work to treat this type of cancer?
</span><span>B) By binding to the active site, the drug prevents the ability of Abl kinase to bind to its substrate.
</span>
Imatinib works against CML by binding close to the ATP binding site of bcr-abl. The binding results to the<span> locking in of the bcr-abl to a closed or self-inhibited conformation and inhibiting the enzyme activity of the protein </span><span>semi-competitively.</span>
Answer:
The organisms homeostasis
Answer:
i think its D because the plant cell runs off the sugar cells
Explanation:
<span>Actually baby Aiden will surely develop a strong secretory Iga antibodies which is passive immunity to most of the types of bacteria and viruses, ie, as a protection against infections, which inturn is a response to the baby to its immune system in the form active immunity.</span>
Answer:
a. add more of the enzyme
Explanation:
Enzymes have specific sites to which their substrates bind during the reaction. These sites are called active sites. When all the active sites of all the enzyme molecules present in a solution are bound to the substrate molecules, the enzyme is said to be saturated with the substrate. Under these conditions, more enzyme molecules are to be added to the solution to increase the reaction rate and to obtain the product at a fast rate. The addition of more enzymes will allow more substrate molecules to occupy the active sites and to be converted into the product/s.
