Homologous or sister chromatids
Answer:
d. Humans and other animal hosts lack peptidoglycan cell walls.
Explanation:
Humans and other animals lack cell walls. Bacterial cells have peptidoglycan cell walls around them. The absence of cell walls in the cells of human and other animal host ensures that these cells are not affected by the drugs that target the peptidoglycan cell walls to kill or slow down the growth of the bacterial pathogen.
This makes the drugs specifically target the bacterial cells only while not affecting the host cells. If a drug would target a component present in cells of both the pathogens and hosts, the host cells would be most likely to be harmed by the drugs.
Answer:
28 units
Explanation:
This disorder follows quantitative inheritance. It is controlled by three genes which do not show the usual dominant-recessive relationship . The six alleles individually contribute to the effect which add up to produce the cumulative phenotype. Dominant allele contributes 6 units of risk whereas recessive allele contributes 2 units of risk.
Individual with genotype AABbCc has four dominant alleles (AABC) and two recessive alleles (bc). So their total risk units =
(6*4) + (2*2) = 24 + 4
= 28 units
As verbalized, the enzyme glucose-6-phosphate dehydrogenase avails red blood cells function felicitously. Ergo, G6PD deficiency can cause hemolytic anemia characterized by having low calibers of red blood cells. The parasite that causes malaria does not survive well in G6PD deficient cells since they appear to have resistance against the disease.
For the first question, I would say a line graph. And the one about the human heart, I need some more information.
