A. show the hierarchy of an organization
D. The tendency of a cell to maintain a stable internal environment.
In cases of acute renal failure, serum creatinine (SCR) and blood urea nitrogen (BUN) incorrectly predict the glomerular filtration rate (GFR). The possibility exists for serum cystatin C (CYS) to be a more accurate GFR marker. In order to examine the sensitivity of SCR, BUN, and CYS in identifying acute renal failure in mice, this study was conducted.
The conclusion of the study was that In mouse models, CYS can be utilized as an accurate and dependable marker for renal function. In comparison to SCR and BUN, CYS is more sensitive and detects kidney impairment early.
<h3>What is
glomerular filtration rate (GFR)?</h3>
A blood test called a glomerular filtration rate (GFR) measures how well your kidneys are functioning. Glomeruli are little filters found in your kidneys. These filters aid in clearing the blood of waste and extra fluid. How much blood flows through these filters each minute is determined by a GFR test.
To learn more about glomerular filtration rate (GFR) with the help of given link:
brainly.com/question/13064727
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<h2>Fatty acid oxidation </h2>
Explanation:
- Activation of fatty acids (palmitate) occurs in the cytoplasm where fatty acids are activated to fatty acyl CoA, reaction catalysed by an enzyme called fatty acyl CoA synthetase
- A specialized carnitine carrier system catalyze transportation of activated fatty acid from cytoplasm to matrix of mitochondria, where carnitine system consists of three proteins:
- Carnitine acyl transferase I located in outer membrane of mitochondria catalyze transfer of carnitine to fatty acyl CoA and produce fatty acyl carnitine
- Carnitine translocase facilitate passive diffusion of fatty acyl carnitine from inter membrane space to matrix located in inner membrane
- Carnitine acyl transferase III located in inner mitochondrial membrane at matrix phase catalyze transfer of CoA to fatty acyl carnitine and regenerate fatty acyl CoA
Hence, the cytosolic and mitochondrial pools of CoA are thus kept separate, and no radioactive CoA from the cytosolic pool enters the mitochondria
