Im confused about the question can you explain
I'm not sure of the options listed but: clients typically are obese and clients refrain from purging behaviors.
Explanation:
of the <em><u>46</u></em><em> </em>chromosomes in the nuclei of every diploid human somatic cell, <em><u>23</u></em> chromosomes were contributed by the mother in the <em><u>egg (ovum)</u></em> cell and the other <em><u>23</u></em> chromosomes come from the father’s <em><u>sperm cell. </u></em>
Answer;
The amnion
Explanation;
-The amnion is a tough, thin membrane that surrounds a developing fetus in mammals, reptiles, and birds. It is the first of the three cavities (amnion, chorion and yolk sac) in the embryo and is formed on 8 dpc.
-In the human body, the amnion is the first of three cavities, including the chorion and yolk sac, that work together to keep the developing fetus nourished and protected.
-The amnion provides a protective environment in which the fetus' temperature is regulated and protects the fetus from friction caused by the mother's movements.
Answer:
Bridgham et al. (2006) showed that the interaction between a steroid hormone (aldosterone-M) and its receptor (mineralocorticoid) evolved by Darwinian gradualism. In this work, the authors demonstrated a primitive affinity between the hormone and its receptor that was initially present in chemically similar but more ancient ligands. This result has implications in understanding the association between gene duplication and the evolution of hormone signaling pathways. For example, in invertebrates, this work reinforces the importance of gene duplication in the existing interaction between paralogous glucocorticoid receptors and their receptor mineralocorticoid genes that were derived from duplication (Thornton 2001).
The publications above cited are the following:
J.T. Bridgham, S.M. Carroll, and J.W. Thornton (2006). Evolution of hormone-receptor complexity by molecular exploitation. Science, 312(5770), 97-101.
JW Thornton. Evolution of vertebrate steroid receptors from an ancestral estrogen receptor by ligand exploitation and serial genome expansions, Proc Natl Acad Sci USA (PNAS), 2001, vol. 98 10 (pg. 5671-5676).
