Answer:
The answer is "Option e".
Explanation:
please find the complete question in the attached file.
Its long tail disregards its short tail. Let's assume that even a short neck is a as well as a tail over, which claim, though, the short tails were mixed, shorter, and longer tailed mousses are created. It may also presume that the short mouse parental is always Aa. And we get AA, Aa, Aa, Aa, and Aa situations once their matter and they fall pregnant to both high and short tail mice but we wouldn't get the fat tail mouse unless the tail-mouse were as AA.
we always get two types of lines. It demonstrates there was no uniform AA genera. It is a case of the heterozygous dangerous gene, that can cause a set of identical alleles inside an organism to always be lethal.
One: The cell is able to move material in and out of the cell
Two: It helps the cell maintain necessary conditions for life
Religion because of the religion
A. Consumers cannot produce their own food source so they have to eat other animals.
C. Rhizobia bacteria "prepare" the atmospheric nitrogen and make it useful for the plant.
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There are a variety of points in the transcriptional chain at which it is possible to disrupt protein synthesis in bacteria. Let’s enumerate just a few:
<span>There’s the initial point where DNA is transcribed into mRNA;<span>there’s the point where mRNA binds to the Ribosome complex;</span>there’s the point where tRNA-aminoacyl pair binds to the Ribosome according to the current codon being “read out” in the mRNA;there’s the point where the aminoacid transported by the tRNA is transferred to the growing protein chain; andthere’s the point where the protein synthesis is determined complete, and the Ribosome disengages and releases the newly-synthesized peptide chain.</span>
In each of these stages (and in some other, more subtle phases) there are possible points of disruption and there are specific disruptors; some of which are indicated in the aboveProtein synthesis inhibitor article.
Note, by the way, that the Ribosomes of Prokaryotes (bacteria) and Eukaryotes (cells with nuclei) aren’t identical, and therefore the inhibitors/disruptors that work for one type of cell may not (and usually don’t) work on the other type. That’s why we can take antibiotics targeted at bacteria with little to no fear of them interfering with our eukaryotic cells’ functions.
(This is a simplified, somewhat hand-wavy response. There is a lot more to say, mainly because biological systems are anything but simple. Nevertheless this should be enough to get you started in the general direction.)