Question

You are interested in studying a gene called CFTR because mutations in this gene cause cystic fibrosis. You have made a line of mice that lack the mouse CFTR gene. These mice are unable to clear bacteria from their lungs, so they get lung disease. You put a normal human CFTR gene into some of these mice and discover that the mice with the human gene are able to clear bacteria from their lungs and no longer get lung disease. From this experiment, you can conclude that:_______.
A. The DNA sequences of the mouse CFTR gene and human CFTR gene are identical (0.7%).
B. The amino acid sequences of the mouse CFTR protein and the human CFTR protein are identical (1.1%).
C. The mouse CFTR gene and human CFTR gene encode proteins that can serve a similar function (70.9%).
D. Both answers b and c are true (20.8%).
E. All of the above are true (6.5%).

Answer 1

Answer:

The mouse CFTR gene and human CFTR gene encode proteins that can serve a similar function.

Explanation:

CFTR (Cystic fibrosis transmembrane conductance regulator) is the important membrane protein that plays an important role in the transfer of the chloride channels in the body.

The mutation in CFTR is responsible for cystic fibrosis. As the information given in the question, it might be possible that the gene CFTR plays the quite similar role in both human and mice. These gene might not necessary to have same DNA sequence but perform the similar function.

Thus, the correct answer is option (C).