The answer is: <span>larger horses have longer legs.
</span>The ancestors of horses lived in the forest, where they could hide from the predators. But, with the first steppes, they could be spotted by the predators. Horses had to adapt in order to survive. Thus, their legs became longer through time so horses could run faster to escape predators.<span>
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Answer:
rhizobia is an important nitrogen fixing bacteria found in the soil to help grwo plants like legumes. rhizobia enter into a symbiotic relationship withe legumes
Explanation:
rhizobia and legumes coexist by entering into a symbiotic association. rhizobia is a bacteria that fixes nitrogen molecules for the legumes and in return the legumes provide the bacteria with food and nutrition.
If rhizobia becomes extinct in the near future it would be difficult for plants like legumes to grow as they wouldn't get sufficient nitrogen and nitrogen is an important nutrition for the plant to grow and develop. Thus it can cause rapid depletion or death of plants that need rhizobia to fix nitrogen molecules for them.
Answer:
It increased their life expectaion.
Explanation:
It says it on the graph.
Answer:
Viruses replicate only inside host cells.
Explanation:
<u>Viruses are not used as evidence to support the cell theory because they are not considered a cell and cannot carry out vital life processes without getting into host cells. </u>
According to the cell theory, the cell is the basic unit of life, all living organisms are made up of cells and cells arose from preexisting cells. A virus that is not in a host cell is just a piece of DNA/RNA surrounded by a protein coat and is not capable of carrying out any life process for it to be considered living.
<em>Viruses are only able to reproduce and multiply when they get access to a living host. They use the genetic mechanisms of the host to replicate their DNA/RNA using the lytic or lysogenic cycle. Hence, they are not used to support the cell theory. </em>
When looking at a Neuromuscular Junction (NMJ), we know that neurotransmitters (NT) are released from the presynaptic cell and they then bind to the receptors that are located on the postsynaptic cell - this causes the effect of the NT being released.
So we are told that NT are still being released, however they are not having an effect. This would mean that they are probably being blocked by something - in this case, it seems that the neurotoxin is the culprit in the blocking of these receptors.
Therefore, if the NT cannot bind to the receptors on the postsynaptic cell, they are not going to have any effect, no matter how much NT is being released.
So the answer in this case is: The neurotoxin is most likely C) Blocking the receptors on the postsynaptic cell.
