Answer:
"I will take three nitroglycerin tablets 5 minutes apart, and if I do not have any relief I will seek emergency care immediately."
Explanation:
sublingual nitroglycerin is prescribed to clients with stable or unstable angina to relieve chest pain symptoms.
The client is advised to take up to three tablets of sublingual nitroglycerin 5 minutes apart for relieve of chest pain and if there's no relieve to seek emergency medical care immediately.
P.S: it is advised to take few doses as necessary to relieve pain at the first sign of pain
Answer:
Explanation:
Well the question was a little unclear, but for disaccharides:
(alpha) Glucose + (alpha) Glucose = Maltose
(alpha) Glucose + Fructose = Sucrose
(alpha) Glucose + Galactose = Lactose
The energy released is used for assembly of actin filament with myosin head. when myosin is attached to ATP its heads cannot bind to actin and therefore muscle will remain in relaxed form. However during muscle contraction an enzyme referred to as ATPase hydrolyses ATP to ADP and organic phosphate in the process releasing energy. The energy released changes the position of head of myosin which facilatate its binding with actin. Myosin moves towards m-line dragging actin along with it reducing the length of sarcomere leading to muscle contraction.
Answer:
d. All of the above
Explanation:
Wildfires are useful in controlling the ecosystem disturbed due to some diseases and insects.
With the help of wildfire, there will be a temporary loss in some region but it will lead to extraordinary growth.
Wildfire improves the soil quality by decomposing the organic material and renewing the soil nutrients.
There are chances of habitat loss but it will increase the production of food grazers.
Certain plants can be reproduced through wildfire due to cracking their seeds.
Answer:
Muscle contraction function.
Explanation:
The nerve endings possess synaptic acetylcholine vesicles ready to be released. The action potential depolarizes the presynaptic terminal and increases the concentration of axoplasmic calcium; Acetylcholine molecules are thus released, so that the concentration of the neurotransmitter at postsynaptic (nicotinic) receptors is temporarily increased. This is followed by post-synaptic membrane depolarization, muscle membrane action potential with increased rnioplasmic calcium concentration, and finally muscle contraction. Acetylcholine is hydrolyzed by acetylcholinesterase and resynaptic at the presynaptic level by cholinecetyltransferase. The etiopathogenesis of myasthenia gravis is autoimmune and there are antibodies against acetylcholine receptors that circulate in the blood, as well as a decrease in the number of receptors on the motor plates, that is, it is produced by the postsynaptic blockage of the myoneural plaque, that generates fatigue and localized or generalized muscle weakness that is characterized by the worsening of the contractile force of the muscle.
